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Hexamethonium

A systematic nomenclature for nAChRs has yet to evolve. An N nomenclature describes receptors present ia muscle as N. These are activated by phenyltrimethylammonium (PTMA) (15) and blocked by t5 -tubocurariae (16) and a-bungarotoxiu (a-BgT) (17). N2 receptors are present ia ganglia and are activated by l,l-dimethyl-4-phenylpipera2inium (DMPP) (18) and blocked by trimethaphan (19) and bis-quatemary agents, with hexamethonium (20) being the most potent. [Pg.521]

Structural Formula (CH3)3N (CH2)6N (CH3)3-2Br Chemical Abstracts Registry No. 60-26-4 (Hexamethonium)... [Pg.761]

Hexamethonium bromide Hexocyclium methyl sulfate Ipronidazole Mefruside... [Pg.1630]

Cf,H f,N2 124-09-4) see Hexamethonium chloride Hexcarbaeholine bromide hexamethylenediamine dihydrochloride C(,H,nCl2N2 6055-52-3) see Chlorhexidine 2,2"-(hexamethylenediiniino)bis[3, 4 -dihydroxyaceto-phenone]... [Pg.2392]

Drugs that block the nicotinic receptors on autonomic ganglia, such as hexamethonium, probably do so by actually blocking the Na+ ion channel rather than the receptor. Generally these receptors appear to resemble the central ones more than those at the neuromuscular junction and dihydro-jS-erythroidine is one drug that it is an effective antagonist in both ganglia and the CNS. [Pg.130]

Potential oscillation was measured in the presence of cholinergic agents (acetylcholine chloride, carbamylcholine chloride, carbamyl- d-methylcholine chloride, and acetyl-/6-methylcholine chloride) and anticholinergic agents (tetramethylammonium chloride, tetra-ethylammonium chloride, succinylcholine chloride, hexamethonium chloride, scopolamine hydrobromide, atropine sulfate, homatropine hydrochloride, and tubocurarine chloride)... [Pg.714]

FIG. 18 Chemical structures of (a) acetylcholine chloride, (b) carbamylcholine chloride, (c) carba-myl-y8-methylcholine chloride, (d) acetyl-/i-methylcholine chloride, (e) tetramethylammonium chloride, (f) tetraethylamonium chloride, (g) succinylcholine chloride, (h) hexamethonium chloride, (i) scopolamine hydrobromide, 0 atropine sulfate, (k) homatropine hydrochloride, and (1) tubocurar-ine chloride. [Pg.716]

Nicotine increased DA levels both in vivo11,193 and in vitro. 94 196 Nicotine197 and its metabolites198 were found to both release and inhibit the reuptake of DA in rat brain slices, with uptake inhibition occurring at a lower concentration than that required for DA release. In addition, the (-) isomer was more potent than the (+) isomer.197 However, the effects of nicotine upon DA release and uptake were only apparent when brain slices were utilized because nicotine was unable to affect DA when a synaptosomal preparation was utilized.197 These results indicate that nicotine exerts its effects upon the DAT indirectly, most likely via nicotine acetylcholine receptors. This finding was supported by the results of Yamashita et al.199 in which the effect of nicotine on DA uptake was examined in PC 12 and COS cells transfected with rat DAT cDNA. Nicotine inhibited DA uptake in PC 12 cells that possess a nicotine acetylcholine receptor. This effect was blocked by the nicotinic antagonists hexamethonium and mecamylamine. Additionally, nicotine did not influence DA uptake in COS cells, which lack nicotinic acetylcholine receptors. [Pg.8]

Ti-ZSM-48 was prepared by the dissolved titanium method using fumed silica (Cabosil), TBOT, H202, and diaminooctane (309-310). Ti-ZSM-48 was also prepared using hexamethonium hydroxide base and by the pre-hydrolysis method (311). [Pg.167]

Absorption of quaternary compounds (e.g., hexamethonium, amiloride) are decreased by food. [Pg.465]

Guinea pig vas deferens Muscarinic nicotinic or Muscarinic Methacholine/atropine Methacholine/hexamethonium Leach, 1956... [Pg.754]

Rat serosal strip Nicotinic Methacholine/hexamethonium Khayyal, et. al., 1974... [Pg.754]

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Hexamethonium bromid

Hexamethonium bromide

Hexamethonium chlorid

Hexamethonium nicotinic receptor antagonist

Hexamethonium species

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