Heart electrocardiogram

Doridosine. Doridosine, AJ -methyhsoguanosine, (35) was isolated from the dorid nudibranchs of Anisodoris nobilis and the sponge, Tedania (106,107). The injection of (35) into the saphenous vein of anesthetized rats produces hypotension and bradycardia almost immediately. The observed changes in the electrocardiograms are minor and indicate Httie interference with conduction of the impulse within the heart (see Cardiovascularagents). 

An arrhythmia may occur as a result of heart disease or from a disorder that affects cardiovascular function. Conditions such as emotional stress, hypoxia, and electrolyte imbalance also may trigger an arrhythmia An electrocardiogram (ECG) provides a record of the electrical activity of the heart. Careful interpretation of the ECG along with a thorough physical assessment is necessary to determine the cause and type of arrhythmia The goal of antiarrhythmic drug therapy is to restore normal cardiac function and to prevent life-threatening arrhythmias. 

There are a few absolute contraindications for deep peeling, mainly physical or mental instability. During pregnancy and lactation any cosmetic intervention is considered undesirable. All patients are required to perform electrocardiogram and complete blood count prior to the procedure. Any heart disease requires special precautions and it is always recommended to work in cooperation with the patient s cardiologist. 

BP, blood pressure BUN, blood urea nitrate CBC, complete blood cell count ECC, electrocardiogram HF, heart failure HR, heart rate K+, potassium SCr, serum creatinine SOB, shortness of breath. 

BNP, B-type natriuretic peptide BP, blood pressure ECC, electrocardiogram HR, heart rate. 

ACE, angiotensin-converting enzyme aPTT, activated partial thromboplastin time ARB, angiotensin receptor blocker BP, blood pressure CBC, complete blood count ECC, electrocardiogram HR, heart rate INR, International Normalized Ratio RR, respiratory rate SCr, serum creatinine, TTP, thrombotic thrombocytopenic purpura. 

The electrocardiogram (ECG) is a non-invasive means of measuring the electrical activity of the heart. The relationship... 

The only way to determine if a comatose patient has SE is by EEG. EEG monitoring should be used for patients who remain unconscious after initial antiepileptic treatment, and for those who received a long-acting paralytic agent or require prolonged therapy for RSE. Treatment should never be delayed while awaiting EEG results. An electrocardiogram (ECG) should be obtained to rule out cardiac dysfunction when hypotension or an abnormal heart rate is observed. 

BP, blood pressure Cl, continuous infusion CT, computed tomography ECG, electrocardiogram EEG, electroencephalography HR, heart rate ICU, intensive care unit IV, intravenous IVP, intravenous push OTC, over the counter MRI, magnetic resonance imaging PE, phenytoin equivalents PR, per rectum RR, respiratory rate T, temperature. 

Electrocardiogram A noninvasive method of recording the electrical activity of the heart. 

Figure 13.4 Electrocardiogram. The electrocardiogram (ECG) is a measure of the overall electrical activity of the heart. The P wave is caused by atrial depolarization, the QRS complex is caused by ventricular depolarization, and the T wave is caused by ventricular repolarization.

Effects No effects on tested parameters of blood pressure, heart rate, electrocardiogram (EKG) rhythms, or lung peak expiratory flow. 

The cardiac sensitization test is based on the observation that some halocarbons make the mammalian heart abnormally sensitive to epinephrine, resulting in ectopic beats and/or ventricular fibrillation, which may result in death. Effects are monitored with electrocardiograms (EKG). The dose of administered epinephrine results in blood levels that may be approximately ten times endogenous levels and is close to the threshold for inducing cardiac effects in the absence of the test chemical. 

Aytemir, K., Maarouf, N., Gallagher, M. M., Yap, Y.G., Waktare, J.E. and Malik, M. (1999) Comparison of formulae for heart rate correction of QT interval in exercise electrocardiograms. Pace-Pacing and Clinical Electrophysiology, 22, 1397—... 

Van de Water, A., Verheyen, J., Xhonneux, R. and Reneman, R.S. (1989) An improved method to correct the QT interval of the electrocardiogram for changes in heart rate. Journal of Pharmacological Methods, 22, 207-217. 

Grabowski CT. 1983b. The electrocardiogram of fetal and newborn rats and dysrhythmias induced by toxic exposure. In Abnormal functional development of the heart, lungs and kidneys Approches to functional teratology. New York, NY Alan R Liss, Inc., 185-206. 

Atropine s peripheral effects on heart rate (Fig. 63) and blood pressure (not shown) are substantial and very rapid in onset, peaking at about 30-60 minutes. In this graph, baseline heart rate is shown as zero. Maximum heart rate at the ID50 is thus about 125 (60 + 65). It remains at this level for about 3 hours and returns to normal at about 9 hours. At the ID50, minor changes in the electrocardiogram were noted in a study by Hayes et al.. These changes rapidly revert to normal as HR declines. 

---