Hamsters, cardiomyopathic

Denovan-Wright EM, Ferrier GR, Robertson HA, Hewlett SE. 2000. Increased expression of the gene for a-interferon inducible protein in cardiomyopathic hamster heart. Bio-chem Biophys Res Comm 267 103. [Pg.384]

In cardiomyopathic hamsters Luque et al. [1994] stained for Cx43 using confocal microscopy and found that some of the cardiomyocytes stain normally but others stain diffusely, with a pixel intensity distribution of the confocal images showing a 90% increase in the number of pixels and a 60% decrease in pixel intensity in the cardiomyopathic hearts as compared to control hearts. Thus, Cx43 seemed to be present in the cells but did not become localized on the membranes as in normal cells. [Pg.82]

Luque EA, Veenstra RD, Beyer EC, Lemanski LF Localization and distribution of gap junctions in normal and cardiomyopathic hamster heart. J Morphol 1994 222 203-213. [Pg.130]

H. Kitabatake, A. Enhanced expression of -adrenergic receptor kinase 1 in the hearts of cardiomyopathic Syrian hamsters, BIO53.58. Biochem. Biophys. Res. Commun., 219, 26-30 (1996)... [Pg.105]

Ikeda, Y., Gu, Y., Iwanaga, Y. et al. (2002). Restoration of deficient membrane proteins in the cardiomyopathic hamster by in vivo cardiac gene transfer. Circulation 105(4), 502-508. [Pg.240]

Bastien NR, Juneau AY Ouellette J, Lambert C. Chronic ATI receptor blockade and angiotensin-converting enzyme (ACE) inhibition in (CHF 146) cardiomyopathic hamsters effects on cardiac hypertrophy and survival, Cardiovasc Res 1999 43 77-85. [Pg.461]

Cai JJ, Yu H, Lee HC. 1993. Developmental changes of protein kinase C and Gs alpha in hypertrophic cardiomyopathic hamster hearts. J Lab Clin Med 122 533-541. [Pg.22]

Horackova, M., Beresewicz, A., Rowden, G., and Wilkinson, M. 1991. Neurohumoral regulation of excitation-contraction coupling in ventricular myocytes from cardiomyopathic hamsters. Car-diovasc. Res. 25 1023-1034. [Pg.45]

Ikegaya, T., Kobayashi, A., Hong, R.B., Masuda, H., Kaneko, M., and Noboru, Y. 1992. Stimulatory guanine nucleotide-binding protein and adenylate cyclase activities in Bio 14.6 cardiomyopathic hamsters at the hypertrophic stage. Mol. Cell. Biochem. 110 83-90. [Pg.45]

Sethi, R., Bector, N., Takeda, N., Nagano, M., Jasmin, G., and Dhalla, N.S. 1994. Alterations in G-proteins in congestive heart failure in cardiomyopathic (UM-X7.1) hamsters. Mol. Cell. Biochem. 140 163-170. [Pg.48]

Witte, K., Olbrich, H.G., Langer, L., and Lemmer, B. 1993. Uncoupling of Pj-adrenoceptors from cardiac adenylyl cyclase in cardiomyopathic and control hamsters. Eur. J. Pharmacol. 247 215-218. [Pg.49]

Nishizawa, T., Iwase, M., Kanazawa, H., Ichihara, S., Ichihara, G., Nagata, K., Obata, K., Kitaichi, K., Yokoi, T., Watanabe, M., Tsunematsu, T., Ishikawa, Y., Murohara, T., and Yokota, M. 2004. Serial alterations of beta-adrenergic signaling in dilated cardiomyopathic hamsters possible role of myocardial oxidative stress. Circ. J. 68 1051-1060. [Pg.174]

Dixon, I.M.C., Hao, J., Reid, N.L., and Roth, M. 2000. Effect of chronic ATI receptor blockade on cardiac smad overexpression in hereditary cardiomyopathic hamsters. Cardiovasc. Res. 46 286-297. [Pg.261]

Takagi C, Urasawa K, Yoshida I, et al. Enhanced GRK5 expression in the hearts of cardiomyopathic hamsters, J2N-k. Biochem Biophys Res Commun 1999 262 206-210. [Pg.337]

Sakata Y. Tissue factors contributing to cardiac hypertrophy in cardiomyopathic hamsters (BI014.6) involvement of transforming growth factor-pi and tissue renin-angiotensin system in the progression of cardiac hypertrophy. Hokkaido Igaku Zasshi. 68 (1993) 18-28. [Pg.321]

Ziegelhoffer A., Tappia P.S., Mesaeli N., Sahi N., Dhalla N.S., Panagia V. Low level of sarcolemmal phosphatidylinositol 4,5-bisphosphate in cardiomyopathic hamster (UM-X7.1) heart. Cardiovasc. Res. 49 (2001) 118-126. [Pg.324]

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