Exports, lead

Ausfuhr,/. export(ation) elimination. ausfUhrbar, a. practicable, feasible exportable. Ausfuhrbewilligung, /. export license. ausfUhren, v.t. carry out, perform, execute amplify, explain clean, purge evacuate excrete export lead out erect Metal.) assay. 

Exports of lead metal increased from 55,500 metric tons in 1990 to 94,400 metric tons in 1991, then fell to 44,000 metric tons in 1996 and 37,400 metric tons in 1997. In 1997, the U.S. exported lead metal primarily to South Korea, Canada, United Kingdom, Malaysia, Belgium, and Taiwan. Lead waste and scrap exports, which amounted to 71,900 metric tons in 1990, rose to 104,300 metric tons in 1995, dropped to 85,300 metric tons in 1996, and rose to 88,400 metric tons in 1997. The lead content of exported scrap lead-acid batteries went from 4,800 metric tons in 1990 to 1,400 metric tons in 1995. No later export tonnage figures for scrap lead-acid batteries are available for 1996 because the data were collected by dollar value only. Most exports are in the form of lead-acid batteries or products containing either lead-acid batteries or other applications of lead (Larrabee 1998 Smith 1998). 

Any increase in biomass content to the dryer results in a reduction in ijo surd a rise in CoE (other parameters remaining unchanged), for the obvious reason that more heat is required for drying and Is therefore unavailable for export, leading to a fall in revenue and an increase in dryer capital cost. 

For these three reasons 1 propose to consider microemulsions and their structure also to be the result of a supercritical energy input and entropy export, leading to self-organised dissipative structures. 

A conservative estimate of the total value of the products from the mineral industry is ca 3.9 trillion ia terms of 1992 dollars (4). This estimate does not include the value of products derived from secondary sources such as recycling (qv) or reclamation. Secondary recovery is significant for certain commodities. For example, in 1992 ca 30% of the world steel (qv) production, 46% of the world refined lead output, 15% of the world refined copper (qv) production, and ca 30% of the aluminum (see Aluminumand ALUMINUM alloys) output from the Western world were clearly identified as being derived from scrap. The value of the world mineral commodity export trade in 1992 was ca 616,698 million ia 1992 dollars. This accounted for ca 18% of all commodities exported (4). 

Primary consumers for ferrous scrap are the iron and steel mills and foundries. Minor consumers iaclude ferroalloy producers, copper producers for use ia copper precipitation (see Recycling, nonferrous metals), and the chemical iadustry. The steel iadustry consumes about three-fourths of the total. Scrap consumption for ferroalloy production, copper precipitation, and the chemical iadustry total less than one million t. The United States is the leading exporter of ferrous scrap, exporting almost nine million t ia 1994, valued at about 1.3 biUioa. Total value of domestic scrap purchases and exports ia 1994 was 8 biUioa (2). 

A problem of many sewage treatment works in the UK is that urban drainage is included with domestic sewage in the sewage collection systems. The resulting storm-water discharges, at times of heavy rainfall, lead to continuing phosphorus export to the river. Correction of this problem requires considerable investment in separate systems. 

However, the power factor of the current drawn from the supply will only improve if the power factor of the generated current is less than that of the parallel loads, since the active power drawn from the supply will also be reduced by the amount of actual power generated. In each case increasing the excitation so that the power factor of the current drawn from the supply can improve to unity or become leading can increase the reactive current produced by the synchronous machine within the capabilities of the machine. In this case, the installation becomes a net exporter of lagging reactive current to the supply. Figure 16.8 illustrates these two cases. 

The NHR contains also the conserved Calcineurin docking site, PxlxIT, required for the physical interaction of NEAT and Calcineurin. Dephosphorylation of at least 13 serines residues in the NHR induces a conformational change that exposes the nuclear localization sequences (NLS), allowing the nuclear translocation of NEAT. Rephosphorylation of these residues unmasks the nuclear export sequences that direct transport back to the cytoplasm. Engagement of receptors such as the antigen receptors in T and B cells is coupled to phospholipase C activation and subsequent production of inositol triphosphate. Increased levels of inositol triphosphate lead to the initial release of intracellular stores of calcium. This early increase of calcium induces opening of the plasma membrane calcium-released-activated-calcium (CRAC) channels,... 

In the case of prothrombin and related clotting factors, interruption of the vitamin K cycle leads to the production of nonfunctional, undercarboxylated proteins, which are duly exported from hepatocytes into blood (Thijssen 1995). They are nonfunctional because there is a requirement for the additional carboxyl residues in the clotting process. Ionized carboxyl groups can establish links with negatively charged sites on neighboring phospholipid molecules of cell surfaces via calcium bridges. 

Iproniazid also prevents the reserpine syndrome in rats. Reserpine blocks vesicular uptake of monoamines which, as a consequence, leak from the storage vesicles into the cytosol. Although these monoamines would normally be metabolised by MAO, they are conserved when a MAO inhibitor (MAOI) is present, and so co-administration of reserpine and a MAOI leads to accumulation of monoamines in the neuronal cytosol. It is now known that, when the concentration of cytoplasmic monoamines is increased in this way, they are exported to the synapse on membrane-bound monoamine transporters. The ensuing increase in monoamine transmission, despite the depletion of the vesicular pool, presumably accounts for the effects of iproniazid on the behaviour of reserpine-pretreated rats. 

As described above, because MAO is bound to mitochondrial outer membranes, MAOIs first increase the concentration of monoamines in the neuronal cytosol, followed by a secondary increase in the vesicle-bound transmitter. The enlarged vesicular pool will increase exocytotic release of transmitter, while an increase in cytoplasmic monoamines will both reduce carrier-mediated removal of transmitter from the synapse (because the favourable concentration gradient is reduced) and could even lead to net export of transmitter by the membrane transporter. That MAOIs increase the concentration of extracellular monoamines has been confirmed using intracranial microdialysis (Ferrer and Artigas 1994). 

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