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Ethanol self-administration

The study of the role of DA in ethanol self-administration is affected by problems not dissimilar from those encountered with other drugs of abuse except that, in contrast to the fair consistency in one direction or in the other among studies on psychostimulants and opiates, much disagreement is registered in the case of ethanol. [Pg.341]

An additional problem with these studies might result from the fact that ethanol preference over water, although utilized for investigating the reinforcing properties of ethanol, may not be a correlate of such properties (George, 1990 Cunningham et ah, 1992). [Pg.341]

From these studies it appears that in rats with a high ethanol intake obtained by selective breeding or by the sweet-fading techniques and on a limited access paradigm of operant ethanol self-administration, neuroleptics consistently reduce responding for ethanol. This effect seems specific for ethanol, since responding for water was unimpaired. [Pg.341]

The interpretation of the mechanism of the influence of neuroleptics on ethanol reinforcement and the appraisal of the precise role of DA is complicated by the fact that DA receptor agonists either direct (e.g. bromocriptine) or indirect (e.g. amphetamine), similarly to neuroleptics, reduce ethanol drinking (Samson et al., 1993). [Pg.341]

In conclusion, DA might play a role in ethanol reinforcement although the specificity of this role is unclear. [Pg.342]


Thiele TE, Marsh DJ, Ste Marie L, Bernstein IL, Palmiter RD (1998) Ethanol consumption and resistance are inversely related to neuropeptide Y levels. Nature 396 366-369 Thiele TE, Koh MT, Pedrazzini T (2002) Voluntary alcohol consumption is controlled via the neuropeptide Y Y1 receptor. J Neurosci 22 RC208 Thorsell A, Rimondini R, Heilig M (2002) Blockade of central neuropeptide Y (NPY) Y2 receptors reduces ethanol self-administration in rats. Neurosci Lett 332 1-4 Timmusk T, Palm K, Metsis M (1993) Multiple promoter direct tissue-specific expression of rat BDNF gene. Neuron 10 475-489... [Pg.525]

Cannon, Dale S., and Laura E. Carrell. 1987. "Rat Strain Differences in Ethanol Self-Administration and Taste Aversion." Pharmacology Biochemistry and Behavior 28 57-63. [Pg.94]

Engel SR, Lyons CR, Allan AM. 5-HT3 receptor over-expression decreases ethanol self administration in transgenic mice. Psychopharmacology (Berl) 1998 140(2) 243-248. [Pg.571]

Studies showing a selective delta antagonist-induced decrease in ethanol self-administration ... [Pg.418]

Lyness WH, Smith FL (1992) Influence of dopaminergic and, serotonergic neurons in intravenous ethanol self-administration in the rat. Pharmacol Biochem Behav 2 187-192. [Pg.385]

Rassnick S, Pulvirenti L, Koob GF (1992) Ethanol self-administration in rats is reduced by the administration of dopamine and giutamate receptor antagonists into the nucleus accumbens. Psychopharmacol 709 92-98. [Pg.388]

Rodriguez de Fonseca, F, Roberts, A.J., Bilbao, A., Koob, G.F, Navarro, M. (1999) Cannabinoid receptor antagonist SR141716A decreases operant ethanol self-administration in rats exposed to ethanol-vapor chambers. Acta Pharmacol. Sin. 20, 1109-1114. [Pg.114]

Alvarez-Jaimes L, Polis I, Parsons LH. Regional influence of CBl receptor signaling on ethanol self-administration by rats. Open Neuropsychopharm 2009 2 77-85. [Pg.601]


See other pages where Ethanol self-administration is mentioned: [Pg.11]    [Pg.11]    [Pg.527]    [Pg.74]    [Pg.419]    [Pg.419]    [Pg.421]    [Pg.421]    [Pg.422]    [Pg.422]    [Pg.164]    [Pg.341]    [Pg.72]    [Pg.1]   


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