Cotton poisoning

Cotton Poisoning CoUon poisoning is the term used to describe the body s reaction to a particle of fiber, like cotton, or of some other foreign substance that gets injected into the bloodstream. It usually appears in two to four hours after injection and causes muscle cramps, chills, cold sweats, and nausea. After two or three hours of discomfort, It usually passes away, leaving the victim exhausted. 

This IS my understanding of cotton poisoning after talking to people who have experienced it and after experiencing it myself. My own reaction was different in that it kept recurring I still don t understand it. 

One of my friends said I had cotton poisoning. He said that he had had It before and there was no way of stopping it. It would probably last another hour or two. He went into the bathroom and ran a hot bath for me — that was the only thing that would help, he said. It did help some. I was still cold, but the hot water relieved the backache I kept sweating and throwing up, though, I don t know how long it took, but I was finally able to get out of the tub and go to bed, completely exhausted. 

Carbol Schwefelsaure, /. iPharm.) sulfocarbolic acid, sulfophenic acid (p-phenolsulfonic acid.), -seife, /. carbolic soap. -suifosMure, /. = Carbolschwefelsaure. vergiftung, /. carbolic acid poisoning. wasser, n. aqueous solution of phenol, -watte, /. carbolized cotton wool. 

Twenty-two cases of endosulfan poisoning were reported in people exposed while spraying cotton and rice fields the dermal route of exposure was assumed to be the primary route of exposure (Singh et al. 1992). The assumption was based on the fact that those spraying rice fields, and who suffered cuts over the legs with the sharp leaves on the rice plants exhibited the more severe toxicity. Three out of the 22 cases exhibited tremors and 11 presented convulsions all patients recovered. 

Octamethyl pyrophosphoramide is a colorless oil, completely soluble in water, benzene, acetone, and many other common organic solvents except the paraffinic hydrocarbons. Its hydrolysis rate has not been measured, but it appears stable in the absence of alkali. In England, this systemic insecticide has been used to control aphids on hops. There it has been calculated that only a negligible quantity of the poison ultimately may find its way into the beer made from the hops. Despite calculations of this sort, the use of octamethyl pyrophosphoramide on food or fodder crops in this country is definitely not to be recommended. However, it may prove useful if properly applied to control certain insects, especially those attacking ornamental plants, such as rosebushes, and possibly on the cotton aphid and grape phylloxera. The compound has only recently been made available experimentally. 

The wide use of pesticides in the republic started in 1959 when 30% and 60% mercaptophos started to be used for the first time in the fight against cotton pests. In 1959-61, the number of incidents of poisoning, especially in the first group, was 23% of all incidents over the ten-year period 7% of those poisoned suffered fatalities. 

Most of the compounds of arsenic are toxic when in contact with the skin, when inhaled, or when ingested. As with arsenics cousin phosphorus above it in group 15 of the periodic table, care must be taken when using arsenic. The compound arsenic trioxide (As O ), an excellent weed-killer, is also carcinogenic. Copper acetoarsenite, known as Paris green, is used to spray cotton for boll weevils. A poisonous dose of arsenic as small as 60 miUigrams can be detected within the body by using the Marsh test. 

Nitrocellulose (NC) Main component in smokeless powder, i.e., gun cotton. Nitroglycerin (NG) Explosive material, originally the basis for dynamite. Oleander extract A poison. 

A reentry study 12 hr after application in Arizona cotton, exposing volunteers to methyl parathion, ethyl parathion, or monocrotophos for 5 hr produced no clinical signs of poisoning. However, cholinesterase depression averaged 147., and both ethyl-and methyl-parathion were found in the blood as well as p-nitrophenol (PNP) in the urine. These workers were not judged in jeopardy as cholinesterase depression was less than 307. and the PNP excretion was less than 4 mg following exposure (22). 

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