Bacterial damage

By contrast, in necrosis, severe physical, chemical, or bacterial damage causes a cell membrane to burst, releasing apoptotic mediators (Sect. 13.4.3) and proinflammatory cytokines into the stroma (Sect. 13.2.2). The cytosolic enzymes continue to make lactic acid in the absence of mitochondrial function, making the necrotic environment strongly acidic and activating lysosomal enzymes (Sect. 13.2.1) to digest the released cytosolic contents. Necrosis is discussed in relation to aggressive periodontitis (Chap. 14). 

Bacterial damage to animal fibres is described more often than fungal damage -... 

Archaeological wood degradation patterns are specific for specific burial sites. Wood in the marine reducing environment, a few centimeters deep in the sediment, will show typical bacterial damage and iron salt precipitation. The region of ground contact of terrestrial wood will have the surface... 

Frank, K. (1994) Measures to preserve food and feeds from bacterial damage. Ubersichten zur Tiererndhrung 22 149-163. 

On the contrary, Asahipak GS series columns [ref. 44] have Improved stability against high pressure, such as 100 kg/cm, and high flow rate as represented in Table 6. Also Asahipak columns can be used with methanol to avoid bacterial damage to the columns. Also, after the column has been loaded with biologically complex samples whose components are not fully known, one can later wash the column... 

Any delay before preservation increases the risk of bacterial damage especially to the sensitive grain layer. Due to special prescriptions for slaughter houses bactericides are not normally used. 

Birbir, M., Eryihnaz, S., Ogan, A. Prevention of halophilic bacterial damage on brine cured hide with halocins. J. Soc. Leather. Technol. Chem. 2004, 88(3), 99-104. 

Muragenicuy, genotoxicity Rats, mice, bacterial strains, yeasts Few days Potential to cause mutations, chromosomal damage, and... 

In the very early phases of the acute inflammatory response most of the cells invading the damaged area are polymorphonuclear neutrophils, also denoted as PMNs, which serve as initial line of defense and source of proinflammatory cytokines. These cells, which usually live for 4-5 days, circulate in the blood until they are attracted by chemokines into injured tissues. Whereas physical injury does not recruit many neutrophils, infections with bacteria or fungi elicit a striking neutrophil response. The characteristic pus of a bacterial abscess is composed mainly of apoptotic (apoptosis) and necrotic PMNs. Emigration of neutrophils from the blood starts with a process denoted as margination where neutrophils come to lie at the periphery of flowing blood cells and adhere to endothelial cells (Fig. 1). L-Selectin is expressed... 

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