Thyroid-stimulating hormone and

Cannabimimetics are also shown to affect reproductive and metabolic functions indirectly by hormonal modulation through the hypothalamic and pituitary regulatory centers. They are found to reduce serum levels of the luteinizing hormone, prolactin, growth hormone, and thyroid-stimulating hormone, and to increase corticotropin (Murphy, 1998). 

Up to 30% of patients on maintenance lithium therapy develop transiently elevated serum concentrations of thyroid-stimulating hormone, and 5% to 35% of patients develop a goiter and/or hypothyroidism, which is dose-related and more likely to occur in women. This is managed by adding levothyroxine to the regimen. 

Somatostatin inhibits secretion of growth hormone and other hormones such as prolactin from the anterior pituitary and is widely distributed in the brain in interneurons and projection pathways. All parts of the cortex contain local circuit somatostatin positive neurons, concentrated in layers V and VI, as does the amygdala and striatum. The nucleus accumbens and adjacent ventral putamen and caudate—designated limbic striatum—have particularly high concentrations of fibres. By contrast, TRH which regulates release of thyroid stimulating hormone and prolactin by the pituitary is generally confined to nuclei in and around the hypothalamus. 

Two additional recombinant hormones have recently gained marketing approval thyroid-stimulating hormone and calcitonin. 

Table 37-3 Diagnostic Uses of Thyroid-Stimulating Hormone and Adrenocorticotropin.

Glucocorticoids given chronically suppress the pituitary release of ACTH, growth hormone, thyroid-stimulating hormone, and luteinizing hormone. 

Previous diagnostic evaluation had included an electroencephalogram, brain scan, and chromosomal studies, which were unremarkable. Also normal were the plasma levels of electrolytes, calcium, phosphorus, magnesium, bilirubin, thyroxine, thyroid-stimulating hormone, and growth... 

Thyrotropinreleasing factor TRF receptor 3-residue peptide amide Stimulates thyroid-stimulating hormone and prolactin secretion Solid-phase A -Boc chemistry... 

STN24 Welch, C. and Nadkami, S. (1991). Evaluation of the Stratus II immunoassay system for thyroxine, thyroid stimulating hormone, and human chorionic gonadotropin. Clin. Chem. 37, 1041-1042, Abstr. 627. 

Ismail AAA, Walker PL, Barth JH, Lewandowski KC, lones R, Burr WA. Wrong biochemistry results two case reports and observational study in 5310 patients on potentially misleading thyroid-stimulating hormone and gonadotropin immunoassay results. Clin Chem 2002 48 2023-9. 

Condhffe P, Weintraub B. Pituitary thyroid-stimulating hormone and other thyroid-stimulating substances. In Gray C, James V, eds. Hormones in blood. Vol 3. London Academic, 1979 499-574. 

C7. Condliffe, P. G., and Robbins, J., Pituitary thyroid-stimulating hormone and other... 

E5. Ensor, J. M., and Munro, D. S., A comparison of the influence of thyroid-stimulating hormone and cyclic 3, 5 -adenosine monophosphate on the in vitro release of radio-iodine from the thyroid glands of mice. J. Endocrinol. 38, XXViii (1967). 

Licastro F, Moccheneciani E, Zammotti M, Arena G, Masi M and Eabris N (1992) Zinc affects the metabolism of thxroid hormones in children with Down s sindrome normalisation of thyroid, stimulating hormone and of triiodothyronine plasmic levels by dietary zinc supplementation. Int J Neurosa 65 259-268. 

Hood, A., M. L. Allen, Y. Liu, J. Liu, and C. D. Klaassen. 2003. Induction of T(4) UDP-GT activity, serum thyroid stimulating hormone, and thyroid follicular cell proliferation in mice treated with microsomal enzyme inducers. Toxicology and Applied Pharmacology 188 6-13. 

Thyroid-stimulating hormone and thyroglobulin (difficulties in obtaining blood samples, difficult to interpret, high cost). 

B8. Bitensky, L., Alaghband-Zadeh, J., and Chayen, J., Studies on the thyroid stimulating hormone and the long-acting thyroid stimulating hormone. Clin. Endocrinol. 3,363-374 (1974). 

H4. Hall, R., The immunoassay of thyroid-stimulating hormone and its clinical applications. Clin. Endocrinol. 1, 115-125 (1972). 

We produce all kinds of hormones, but there are two major classes peptides (typical examples are insulin and thyroid-stimulating hormone), and steroids (which include the sex hormones estrogen, progesterone, and testosterone, as well as the stress hormone, cortisol). 

Lithium accumulates in the thyroid gland and blocks the release of the thyroid hormones by thyroid-stimulating hormone, and can therefore cause clinical hypothyroidism. " The prevalence of hypothyroidism may be higher in women, in middle-age, and in countries with a higher level of nutritional iodine. Potassium iodide temporarily prevents the production of thyroid hormones but, as time goes on, synthesis recommences. Thus, both lithium and iodide ions can depress the production or release of the hormones and therefore have additive hypothyroid effects. 

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