Random cells

The scoring of foci was carried out according to the recommended guidelines. Only foci considered as positive (type III), showing deeply basophilic, dense multilayering of cells, random cell orientation at all parts of the focus edge, invasion into the surrounding contact-inhibited monolayer, and domination of spindle-shaped cells, were counted. 

Trigger programmed cell death (apoptosis) as opposed to random cell death (necrosis). This allows sub-lethal doses of photosensitiser, preventing damage and inflammatory response in healthy tissue. 

Until now, the theoretical discussion has focused on monodisperse two-dimensional model systems. However, some studies have been performed on polydisperse systems, notably by Weaire et al. [68-72] The evolution of a soap froth of random cell sizes and shapes, known as a Voronoi network, was simulated by computer [68] (Fig. 7). The condition that three films must always meet at angles of 120° was again used. Cells with more than six sides were found... 

Then there are flexible linear polymers which curl up in solution to give a random cell. If the chain is stiff, such as in cellulose or in DNA, the coil becomes highly expanded. 

Akashah et al. optimum feed, 118.119 Albright random cells, 542 AMSYM program, 171,192 ASPEN system, 163 ASPENPlus system, 163, 177,179 187 192... 

The sites of targeting of IL on hypoxic cardiocytes was visualized by fluorescent microscopy, using rhoda-mine labeled antimyosin immunoliposomes. Fig. 17 shows that only cells treated with antimyosin-rhoda-mine labeled IL were still confluent in the culture and that almost all cells were labeled with fluoresent liposomes. Those cells treated with rhodamine labeled PL showed extremely sparse number of cells still attached to the culture plates at 24 h of incubation, with essentially no or minimal fluorescence. Confocal microscopic examination of the cultures treated with rhodamine labeled IL showed that the cells still retained their morphology and shape with scattered fluorescent liposomes attached to the cell membranes (Fig. 18A). Those cells treated with rhodamine labeled PL were shrunken and only a few random cells showed some non-specific attachment of fluorescent PL (Fig. 18B). In this study, untreated hypoxic cells were all dead and since there was no fluorescent compounds added in them, no micrographs were obtained. 

TEM studies of thin transverse fiber sections show that the cortical structure of cashmere is considerably different from that of fine wool [296,311,320]. Australian and Chinese cash-mere fibers display both bilateral symmetry and random cell arrangements, not only in cashmere fibers from different samples but also in fibers from the same fleece [296,311], whereas fine wool fiber exhibits bilateral asymmetry only. The variation in cortical structure among fibers from the same cashmere fleece suggests that different mechanisms may be involved in fiber formation. Cashmere cortex is composed predominantly of ortholike and mesolike cells, whereas fine wool is composed predominantly of ortho- and paracortical cells arranged bilaterally. Because of the variations observed, many transverse sections need to be examined before definitive statements can be made about the physical structure of fiber from a given cashmere sample. 

Camel fibers from the same fleece can exhibit both bilateral and random cell arrangements. Similar observations have been made for yak fibers, which tend to consist mainly of ortho- and mesolike cells [296]. Vicuna and guanaco exhibit bilateral structure, whereas llama and alpaca do not [296]. 

In order to distinguish between directed cell migration and increases in random cell motility, and provide a more... 

Strauss, G. H. Non-random cell killing in cryopreservation implications for performance of the battery of leukocyte tests (BLT), I. Toxic and immunotoxic effects. Mutat. Res. 1991, 252, 1-15. 

The model predicts that in Gi cells, which are known to be noninducible for TAT, the synthesis of TAT on preexisting templates should be constitutive. This has been examined [58]. It was found that, in G2 cells which had been previously induced for TAT, the removal of inducer did not result in the repression of TAT synthesis as it did in a population of random cells from the same vessel, and this lack of repression is in direct concurrence with the model [46,58]. In this same experiment the degradation of TAT was identical in the G2 and the random cells [58]. 

There is a second function in HTC cells which similarly does not appear to be induced in early G,. Enhanced degradation of TAT can be effected by a nutritional step down of the cells and requires concomitant protein synthesis, and to a somewhat less extent, RNA synthesis [8]. When synchronized HTC cells are stepped-down in mitosis, enhanced degradation does not commence until about G,(3), but its appearance in populations of random cells is immediate [61]. Whether this restriction of induction affects all inducible macromolecules or specifically only TAT-related functions is not clear. However, it has been reported that immunoglobulin synthesis does not occur in cells located in G2 or in early Gi [62]. 

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