Rabbit alveolar macrophages

Romert, L. and Jenssen, D. (1983). Rabbit alveolar macrophage-mediated mutagenesis of polycyclic aromatic hydrocarbons in V79 Chinese hamster cells. Mutat. Res. Ill, 245-252. 

Robinson AV. 1982. Effect of in vitro exposure to hydrogen sulfide on rabbit alveolar macrophages cultured on gas-permeable membranes. Environ Res 27 491-500. 

Dowell, A. R., L. A. Lohrbauer, D. Hurst, and S. D. Lee. Rabbit alveolar macrophage damage caused by in vivo ozone inhalation. Arch. Environ. Health 21 121-128, 1970. 

TABLE VI. Viability of Rabbit Alveolar Macrophages Exposed to Sub-Micron Particle Filters... 

In studies on rabbit alveolar macrophage cultures, Waters and coworkers (133) presented data suggesting that vanadium oxides may adversely affect pulmonary defense. The cytotoxicity of the oxides studied were directly related to their solubility, i.e., V2O5 > V203 > V02. Ambient vanadium concentrations in urban regions have been reported to correlate with mortality incidence from bronchitis and pneumonia, especially in males (134). Likewise, industrial exposure to airborne manganese has been shown to correlate with increased incidence of bronchitis, caused in part by increased susceptibility to infection (135). 

Nakagawa Y., Kurihara K., Sugiura T., and Waku K. (1985). Heterogeneity in the metabolism of the arachidonoyl molecular species of glycerophospholipids of rabbit alveolar macrophages. The relationship between metabolic activities and chemical structures of the arachidonoyl molecular species. Eur. J. Biochem. 153 263-268. 

Southwick, F.S., and MJ. DiNubile. 1986. Rabbit alveolar macrophages contain a Ca2+-sensitive, 41,000-dalton protein which reversibly blocks the barbed ends of actin filaments but does not sever them. J Biol Chem. 261 14191—5. 

H. Tomoda, Y. Kishimoto, and Y. C. Lee, Temperature effect on endocytosis and exocytosis by rabbit alveolar macrophages, J. Biol. Chem. 264 15445-15450 (1989). 

Rothberger H, McGee MR Generation of coagulation factor V activity by cultured rabbit alveolar macrophages, J Exp Med 1984 160 1880-1890. 

M7. Mahoney, E. M., Khoo, J. C., and Steinberg, D., Lipoprotein lipase secretion by human monocytes and rabbit alveolar macrophages in culture. Proc. Natl. Acad. Sci. U.S.A. 79, 1639-1642 (1982). 

Johansson A, Wiemik A, Jarstrand C, et al. 1986b. Rabbit alveolar macrophages after inhalation of hexa- and trivalent chromium. Environ Res 39 372-385. 

Waters MD, Gardner DE, Arany C, et al. 1975. Metal toxicity for rabbit alveolar macrophages in vitro. Environ Res 9 32-47. 

Shafa, F., Moberly, B.J., Gerhardt, P. (1966). Cytological features of anthrax spores phagocytized in vitro by rabbit alveolar macrophages. J. Infect. Dis. 116 401-13. 

A18. Axline, S. G., Isozymes of acid phosphatase in normal and Calmette-Gu4rin bacillus-induced rabbit alveolar macrophages. J. Exp. Med. 128, 1031-1048 (1968). 

Mammalian defensins 3 P Sttands 3 disulfides Rat, rabbit, guinea pig, human neucrophifs. rabbit alveolar macrophages, human, mouse Paneth cells MCP,NP,HNP,GNCP. rat NP, ciyptidins... 

Triacylglyceroprotein acylhydrolase (EC 3.1.1.34) was secreted by human blood monocytes and New Zealand White rabbit alveolar macrophages cultivated in vitro (Mahoney et al. 1982). 

Concanavalin A and phorbol myristate acetate greatly increased secretion of rabbit alveolar macrophage plasminogen activator, although the higher concentration of 10.0 pg Con A/ml had an adverse effect on viability (Schuyler and Forman 1984). 

Rabbit alveolar macrophages exhibited an apparent discrepancy between the abundance of immu-nospecific cP450 4A protein and no 20-hydroxy-eicosatetraenoic acid production (Zhu et al. 1998). NO generated by alveolar macrophages may auto-inhibit the P450 4A enzymes and production of 20-HETE in these cells. 

Pentoxifylhne (1 mM) stimulated the rate of rabbit alveolar macrophage spreading on glass covers-hps more than twofold (Wang et al. 1996). It reduced superoxide generation induced by phorbol-myristate-acetate by >50 %. Production of TNF-a induced by lipopolysaccharide was suppressed >85%. 

Rabbit alveolar macrophages incubated for 20 h in supplemented Medium 199 with particulate... 

In vitro, amiodarone decreased the surface density of rabbit alveolar macrophage mitochondria and lysosomes while increasing the surface density of inclusion bodies, increased the incorporation of choline into dipalmitoylphosphatidylcholine, modified the distribution of lysosomal enzymes, and did not affect the uptake and processing of diphtheria toxin (10 nM) but inhibited the degradation of surfactant protein A (Baritussio et al. 2001). 

Arachidonic acid release from rabbit alveolar macrophages gained by bronchoalveolar lavage was increased by tarmin in a dose-dependent manner (Ralston and Rohrbach 1994). 

While the number of macrophages recoverable from cultures of rabbit alveolar macrophages exposed to hyperoxia for 72 h was decreased compared with that in normal control cultures exposed to normoxia for similar durations, addition of di-methylthiourea or catalase (EC 1.11.1.6), but not superoxide dismutase (EC 1.15.1.1), increased the number of macrophages recoverable from cultures exposed to hyperoxia (Harada et al. 1983). Alveolar macrophages exposed to hyperoxia in the presence of dimethylthiourea or catdase, but not superoxide dismutase, did not develop ultrastructural abnormalities as vacuolisation and cytoplasmic and nuclear degeneration. 

Rabbit alveolar macrophages and monocytes, human blood monocytes and granulocytes... 

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