Peritoneal dialysis benefits

Seven patients who were in supportive treatment (without need for dialysis) at the time of star fruit ingestion had rruld intoxication presenting hiccups or diarrhea. Six patients improved without dialysis. Time to improve was up to 24 hours in 4 patients, 5 days in another one, and there is no information in one patient. One patient improved after IPD (intermittent peritoneal dialysis) [13,14]. Peritoneal dialysis was not an efficient method of treatment although 1 patient with signs and symptoms of moderate intoxication and 2 with mild intoxication changed from CAPD to IPD (intermittent peritoneal dialysis) and improved [13]. Two patients that remained in CAPD also improved [7, 12]. In one case [13] patient presented diplopia that continued for 6 weeks after improvement of the acute intoxication episode. Patients with severe intoxication did not benefit from peritoneal dialysis treatment [13]. 

Williams PF, Moncrieff N, Marriott J. No benefit in using nystatin prophylaxis against fungal peritonitis in peritoneal dialysis patients. Perit Dial Int 2000 20 352-353. 

Total replacement doses of IV iron dextran have been given as a single dose, diluted in 250 to 1000 mL normal saline or 5% dextrose in water and infused over 4 to 6 hours. A test dose is still required. The ability to give a total dose infusion is a benefit of iron dextran over the other parenteral iron products. Iron dextran is best utilized when smaller frequent doses of sodium ferric gluconate or iron sucrose are impractical, such as with peritoneal dialysis. 

Silicone elastomer catheters employed in peritoneal dialysis have been dip-coated with silver oxide or by using IBAD. These studies demonstrate that the elution of silver was available over periods of time in excess of 9months (Kubey etal. 1995). Although the benefits of the use of silver in medical implants remain arguable, it is evident that its use in such applications is increasing. 

Chronic renal failure patients on hemodialysis and peritoneal dialysis are at risk for thiamine deficiency due to inadequate nutrition in part and possible thiamine loss during the dialysis process. Renal failure patients are often on a diet restricted in protein and potassium, which increases the risk of thiamine deficiency (Masud, 2002 Piccoli et al, 2006). Studies with detailed dietary surveys have shown poor oral intake of thiamine in chronic renal failure patients (Hung et al., 2001). There is no convincing evidence that thiamine levels are significantly altered by either hemodialysis or peritoneal dialysis (Reuler et al, 1985). DeBari et al (1984) measured thiamine levels of granulocytes, erythrocytes and plasma. They found no significant differences in thiamine levels in dialysis patients compared to controls. Further research in this area would benefit chronic renal failure patients and help determine possible need for supplementation of water-soluble vitamins. 

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