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Patient Restraints

We can define restraints as any manual method, physical device, or mechanical device used to restrict the freedom of movement or normal access to one s body. Due to an increasing number of reports of injury and death associated with the incorrect use of patient restraints, the FDA warns health professionals to ensure the safe use of these devices. Restraints can include safety vests, lap belts, wheelchair belts, and body holders. Incorrect use of these devices has involved using the [Pg.331]

We can define restraints as any manual method, physical device, or mechanical device used to restrict the freedom of movanent or normal access to one s body. Due to an increasing number of reports of injury and death associated with the incorrect use of patient restraints, the IDA warns health professionals to ensure the safe use of these devices. Restraints can include safety vests, lap belts, wheelchair belts, and body holders. Incorrect use of these devices can involve using the wrong size for a patient s weight, errors in securing restraints, and inadequate patient monitoring. Such mistakes can result in fractures, bums, and strangulations. We can simply define a restraint as any manual method, physical device, mechanical device, material, or equipment attached or adjacent to a patient or resident s body that restricts freedom of movement or normal access to one s body. Under this functional definition, other devices or facility practices also may meet the [Pg.100]

Safety Bars Used on wheelchairs to prevent falls. [Pg.101]

Soft Belts Similar to seat belts used to prevent falls from beds and wheelchairs. Safety Vest Provides more support in preventing falls from a chair or bed. [Pg.101]

Wrist Restraint A hmb-holding restraint that prevents the patient from removing tubes or bandages. Note Physically check every 15 minutes to ensure circulation. Mitt Restraint This type of restraint restricts finger movanent but permits movement of the arm and wrist. [Pg.101]


The first addend is positive, and measures the direct effect of the rise of the co-payment rate. It expresses the effect of the normative change in the distribution of the financial burden between the two parties, before the user s reaction of restraint in the use of the pharmaceutical is taken into consideration. The other two addends are negative, and quantify the decrease in expenditure caused by the drop in consumption as a reaction to the rise in the price paid. The balance can have either sign, depending on the elasticity of demand and the size of the increase in the co-payment. In the example shown in Figure 7.3, despite a considerable decrease in the quantity consumed, the patient will end up paying more for the drag than before. [Pg.134]

During periods of acute psychosis, some patients exhibit so much muscular activity that they develop muscular destruction with the muscle product myoglobin in urine, which produces acute renal failure (16). Some muscle destruction may be due to involuntary muscle activity induced by the drug, while some may be due to the struggles of the agitated patient. In the latter case, the use of restraints may worsen the situation. [Pg.144]

Additional ADRs linked to diet pills include psychosis myocardial ischemia drug interactions, such as the interaction of fenfluramine with imipramine, fenfluramine with amitriptyline or desipramine, or the toxic reaction between fluoxetine and phentermine and the release of serotonin while inhibiting its reuptake, contributing to hyperserotonin reactions. When the next craze takes hold of patients and their physicians, hopefully physicians and pharmacists will take a more vocal position and recommend restraint, xmtil some proof of efficacy and lack of toxicity is shown for new faddish off-label combinations. [Pg.509]

Diet/Physicai activity The use of acarbose must be viewed by both the physician and patient as a treatment in addition to diet, and not as a substitute for diet or as a convenient mechanism for avoiding dietary restraint. [Pg.286]

Intramuscular injections can be given without siting an intravenous cannula, a difficult procedure in a struggling or uncooperative patient. It is necessary to use adequate restraint and exposure of the injection site so that relevant anatomical landmarks can be identified. Patients should be monitored after they have been sedated, as respiratory or cardiovascular complications can ensue, or the violent behaviour re-emerge. [Pg.506]

There is no evidence that ordinary use of ergotamine for migraine is hazardous in pregnancy. However, most clinicians counsel restraint in the use of the ergot derivatives by pregnant patients. [Pg.366]

As noted at the beginning of this chapter, antipsychotic drugs have had a major impact on psychiatric treatment. First, they have shifted the vast majority of patients from long-term hospitalization to the community. For many patients, this shift has provided a better life under more humane circumstances and in many cases has made possible life without frequent use of physical restraints. For others, the tragedy of an aimless existence is now being played out in the streets of our communities rather than in mental institutions. [Pg.637]

ApoSAA, an acute-phase protein, is produced quickly in mice and men in response to a stress (e.g., endotoxin administration, etiocholanolone injection). The apoSAA concentrations rise from less than 1% to more than 25% of the total HDL protein content, depending on the degree of stress (B25, B26). In man, major changes in plasma concentration with disease have been reported, e.g., 100-fold or 1000-fold decreases in concentration with resolution of an acute illness (R18). There are reports that glucose infusion in a normal subject (M22) and in hospital patients (M23) may modify HDL composition and increase plasma apoSAA in HDL and, in vervet monkeys, chair restraint rapidly induces apoSAA production (P3, P5). In cynomolgus monkeys, apoSAA is cleared rapidly from the circulation, more rapidly than apoC-III2 and much more rapidly than apoA-I (B19). [Pg.255]

The speed with which bacteria acquire resistance to antibiotics cautions restraint about prescribing them too frequently. Between 1983 and 1993, the percentage of patients receiving antibiotics rose from 1.4 to 45. During those years, researchers isolated Eschericia coli annually from patients, and tested the microbes for resistance to five types of fluroquinolones. Between 1983 and 1990, the antibiotics easily killed all 92 E. coli strains tested. However, in the interval from 1991 to 1993, 11 of 40 tested strains (28 percent) were resistant to all five drugs. [Pg.170]

It is therefore an object of the highest importance, to infuse into the minds of these persons [the caregivers], just sentiments, with regard to the poor objects placed under their care to impress upon them, that coercion is only to be considered as a protecting and salutary restraint and to remind them, that the patient is really under the influence of a disease, which deprives him of responsibility and frequently leads him into expressions and conduct the most opposite to his character and natural dispositions, (p. 175)... [Pg.433]

Tuke (1996) warned on more than one occasion that medical treatment and institutional care often worsen the conditions of patients. He found that releasing patients from restraint actually makes them less dangerous. Even as a college student volunteer, I made these same observations and then implemented them more fully as I became a physician and a psychiatrist. [Pg.435]


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