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Nitric oxide exogenous

Datta, S., Patterson, E. H. Siwek, D. F. (1997). Endogenous and exogenous nitric oxide in the pedunculopontine tegmentum induces sleep. Synapse 27, 69-78. [Pg.330]

Maletic, S. D., Dragicevic-Djokovic, L. M., Ognjanovic, B. L. et al. (2004). Effects of exogenous donor of nitric oxide-sodium nitroprusside on energy production of rat reticulocytes. Physiol. Res. 53 (4), 439-47. [Pg.357]

G. Rocchitta, R. Migheli, M.P. Mura, G. Esposito, M.S. Desole, E. Miele, M. Miele, and P.A. Serra, Signalling pathways in the nitric oxide donor-induced dopamine release in the striatum of freely moving rats evidence that exogenous nitric oxide promotes Ca2+ entry through store-operated channels. Brain Res. 1023, 243-252 (2004). [Pg.50]

Dimmeler, S, Ankarcrona, M., Nicotera, P., and Brune, B. (1993). Exogenous nitric oxide (NO) generation or lL-l/3-induced intracellular NO prcxluction stimulates inhibitory auto-ATP-ribosylation of glyceraldehyde-3-phosphate dehydrogenase in RINm5F cells./. Immunol. 150, 2964-2971. [Pg.209]

Hepatocyte Mitochondrial Enzyme Activity after Exposure to Exogenous Nitric Oxide or 18 hr after Induction of Endogenous Nitric Oxide Synthesis Using Cytokines plus Upopolysaccharide ... [Pg.226]

MH, Reid DM, Ralston SH. Defective bone formation and 142. anabolic response to exogenous estrogen in mice with targeted disruption of endothelial nitric oxide synfliase. Endocrinology 2001 142 760-766. 143. [Pg.873]

The authors suggested that in this case the effect of sildenafil may have been reduced by impaired gastrointestinal absorption. They speculated that exogenous nitric oxide inhibits nitric oxide sjmthase activity, with a consequent reduction in pulmonary vascular smooth muscle cGMP concentration. Phosphodiesterase type V inhibitors, such as sildenafil, increase cGMP concentration and ameliorate the rebound effect... [Pg.3133]

A. Nakano, G. S. Liu, G. Heusch, J. M. Downey and M. V. Cohen, Exogenous nitric oxide can trigger a preconditioned state through a free radical mechanism, but endogenous nitric oxide is not a trigger of classical ischemic preconditioning, J Mol Cell Cardiol 32, 1159-1167, (2000). [Pg.112]

Treatment of acute respiratory distress syndrome (ARDS) or acute lung injury may be defined as a condition involving impaired oxygenation. The nonpharmacologic therapies include mechanical ventilation. The pharmacologic therapies include the use of exogenous surfactant, corticosteroids, acetylcysteine (antioxidant), ketoconazole, nitric oxide, eicosanoids and their inhibitors, sodium nitroprus-side (vasodilator), pentoxifylline, antiendotoxin, and anticytokine therapy and antibiotics. [Pg.45]

Feelisch, M. (1991). The biochemical pathways of nitric oxide formation from nitrovasodilators Appropriate choice of exogenous NO donors and aspects of preparation and handling of aqueous NO solutions. /. Cardiovasc. Pharmacol. 17(Suppl. 3), S25-S33. [Pg.13]

Mayer, B., Werner, E. R., Leopold, E., Klatt, P., and Schmidt, K. (1994b). Effects of endogenously produced and exogenously added nitric oxide on purified brain nitric oxide synthase. In Biology of Nitric Oxide (M. Freelisch, R. Busse, and S. Moncada, eds.). Portland Press, Colchester, England. Vol. 4, 1-3. [Pg.260]

TABLE III Decrease in Total Cytochrome P-450 Heme and Total Microsomal Heme by Endogenous or Exogenous Nitric Oxide in Cultured Hepatocytes ... [Pg.282]

TABLE V Effects of Endogenous and Exogenous Nitric Oxide on Activity of Heme-Metabolizing Enzymes ... [Pg.284]


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See also in sourсe #XX -- [ Pg.181 ]




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