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Neuro-genetic systems

The performance of neural networks (NN) can be enhanced by the use of genetic algorithms. There are two different approaches.  [Pg.286]

This approach includes the use of GA in order to (a) generate the weights of a neural network (b) generate the architecture of a neural network and (c) generate both the weights and architecture of a neural network. [Pg.286]

This approach defines the evaluation function of the genetic algorithm as a neural network. The genetic operators are used to train the parameters of the neural network. The evaluation function is the forward stage of the neural network, while the genetic parameters are improved in the back-propagation stage of the neural network. [Pg.286]


In many of the early applications of fuzzy logic, the s and B s in the if-then rules had to be calibrated by cut-and-trial to achieve a desired level of performance. During the past few years, however, the techniques related to the induction of rules from observations have been developed to a point where the calibration of rules—by induction from input-output pairs—can be automated in a wide variety of cases. Particularly effective in this regard are techniques centered on the use of neural network methods and genetic computing for purposes of system identification and optimization. Many of the so-called neuro-fuzzy and fuzzy-genetic systems are of this type. [Pg.382]

For nearly 80% of patients with epilepsy, the underlying etiology is unknown.8 The most common recognized causes of epilepsy are head trauma and stroke. Developmental and genetic defects are the cause of about 5% of cases of epilepsy. Central nervous system (CNS) tumors, central nervous system infections, and neurodegen-erative diseases are other common causes. Other important causes of epilepsy are human immunodeficiency virus infection or neuro-cysticercosis infection, primarily occurring in Latin America. [Pg.444]

Levicar N, Dewey RA, Daley E, Bates TE, Davies D, Kos J, Pilkington GJ, Lah TT (2003) Selective suppression of cathepsin L by antisense cDNA impairs human brain tumor ceU invasion in vitro and promotes apoptosis. Cancer Gene Ther 10 141-151 Levicar N, Strojnik T, Kos J, Dewey RA, Pilkington GJ, Lah TT (2002) Lysosomal enzymes, cathepsins in brain tumour invasion. J Neurooncol 58 21-32 Li F, Ackermann EJ, Bennett CF (1999) Pleiotropic cell-division defects and apoptosis induced by interference with survivin function. Nat Cell Biol 1 461 66 Lopes MBS, VandenBerg SR, Scheithauer BW (1993) The World Health Organization classification of nervous system tumors in experimental neuro-oncology. In AJ Levine and HH Schmidek, eds Molecular Genetics of Nervous System Tumors. Wiley-Liss, New York, NY, pp 1-36... [Pg.819]


See other pages where Neuro-genetic systems is mentioned: [Pg.286]    [Pg.286]    [Pg.53]    [Pg.338]    [Pg.319]    [Pg.331]    [Pg.753]    [Pg.805]    [Pg.521]    [Pg.365]    [Pg.465]    [Pg.307]    [Pg.361]    [Pg.157]    [Pg.75]    [Pg.369]    [Pg.202]    [Pg.1474]    [Pg.82]    [Pg.82]    [Pg.152]   
See also in sourсe #XX -- [ Pg.286 ]




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