Nausea clinical presentation

The clinical presentation of nausea and vomiting is given in Table 27-4. Nausea and vomiting may be classified as either simple or complex. 

Hypertensive encephalopathy is a classic feature of malignanthypertension. Its clinical presentation consists of severe headache, mental confusion, and apprehension. Blurred vision, nausea and vomiting, and focal neurologic deficits are common. If untreated, the syndrome may progress over a period of 12-48 hours to convulsions, stupor, coma, and even death. 

The clinical presentations included a generalized rash or urticaria sometimes accompanied by nausea, malaise, vomiting, fever, arthralgias, and angioedema. 

T-2 is one of the more stable toxins, retaining its bioactivity even when heated to high temperatures. High concentrations of sodium hydroxide and sodium hypochlorite are required to detoxify it. Aerosol toxicities are generally too low to make this class of toxins useful to an aggressor as an MCBW as defined in Figure 30-1 however, unlike most toxins, these are dermally active. Clinical presentation includes nausea, vomiting, weakness, low blood pressure, and burns in exposed areas. 

III. Clinical presentation. After acute oral overdose, most agents cause only nausea, vomiting, and diarrhea. Specific features of toxicity are described in Table 11-4. 

III. Clinical presentation. Death is rare. Acute oral overdose usually causes nausea... 

III. Clinical presentation. Shortly after acute ingestion, nausea and vomiting occur, followed by paresthesias of the tongue, lips, and face confusion tremor obtundation coma seizures and respiratory depression. Because chlorinated hydrocarbons are highly lipid soluble, the duration of toxicity may be prolonged. 

III. Clinical presentation. Abmpt onset of profound toxic effects shortly after exposure is the hallmark of cyanide poisoning. Symptoms include headache, nausea, dyspnea, and confusion. Syncope, seizures, coma, agonal respirations, and cardiovascular collapse ensue rapidly after heavy exposure. 

II. Toxic dose. Inhalation or ingestion of as little as 1 mg of fluoroacetate is sufficient to cause serious toxicity. Death is likely after ingestion of mote than 5 mg/kg. Clinical presentation. After a delay of minutes to several hours (in one report coma was delayed 36 hours), manifestations of diffuse cellular poisoning become apparent nausea, vomiting, diarrhea, metabolic acidosis, renal failure, agitation, confusion, seizures, coma, respiratory arrest, pulmonary edema, and ventricular arrhythmias may occur. One case series reported a high incidence of hypocalcemia and hypokalemia. 

III. Clinical presentation. Acute ingestion usually causes prompt nausea and vomiting. Both compounds are volatile, and inhalation of vapors may cause eye, nose, and throat Irritation. 

III. Clinical presentation. In general, patients with NSAID overdose are asymptomatic or have mild gastrointestinal upset (nausea, vomiting, abdominal pain, sometimes hematemesis). Occasionally patients exhibit drowsiness, lethargy, ataxia, nystagmus, tinnitus, and disorientation. 

III. Clinical presentation. Initial symptoms include nausea and vomiting. Occasionally, confusion, stupor, and coma occur after several hours. After a delay of several hours to days, irreversible autonomic neuropathy, peripheral neuropathy, and diabetes may occur. 

III. Clinical presentation. Acute overdose may cause headache, nausea, dizziness, weakness, syncope, orthostatic hypotension, warm flushed skin, and palpitations. Lethargy and ataxia may occur in children. Severe hypotension may result in cerebral and myocardial ischemia and acute renal failure. First-time users of alpha-1 blockers may experience syncope after therapeutic dosing. 

Diagnosis is based on clinical symptoms that include periumbilical or right lower quadrant (RLQ) pain, vomiting, nausea, fever and leucocytosis. Nevertheless, more than 30% of cases of appendicitis have atypical clinical presentations (Lewis et al. 1975), particularly in small children and in cases of unusual appendiceal location (Rothrock et al. 1991 Poole 1990). In addition, the presentation signs and symptoms of many nonsurgical condi-... 

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