Marijuana ethanol

Anti-androgens and hormones 5a-Reductase inhibitors Progesterone and estrogen Recreational drugs Ethanol Cocaine Marijuana... 

Epstein-Barr virus Human T-lymphocyte virus (HTFV-1 and HTFV-2) Social Habits Cigarette smoking Maternal marijuana use Maternal ethanol use... 

The dopaminergic system plays a role in the abuse liability for some, if not most, drugs. The stimulants — opiates, marijuana, nicotine, and ethanol — all interact directly or indirectly with... 

Longitudinal studies of substance use have supported the existence of sequential stages of substance use, beginning with the use of licit substances, such as tobacco and ethanol, and progressing to illicit substances, particularly marijuana (Kandel et ah, 1992). 

Foltin, R.W. et al., Behavioral effects of cocaine alone and in combination with ethanol or marijuana in humans, Drug Alcohol Depend., 32, 93, 1993. 

Heishman, S.J., Singleton, E.G., and Crouch, D.J., Laboratory validation study of Drug Evaluation and Classification program ethanol, cocaine, and marijuana, J. Anal. Toxicol., 20, 468, 1996. 

All the subjects received each of the treatments. Neither the subjects nor the technician knew the identity of the treatment at the time of the experiment. The treatments were randomly presented a minimum of 48 hr apart. On study days subjects swallowed three opaque capsules, drank a large cold tonic drink (480 ml, in 15 min) with 2 ml 95% ethanol floating on top, and smoked a cigarette (either marijuana or placebo) according to a paced puffing procedure 8 puffs per cigarette, 20-s... 

Age, tobacco, marijuana, cirrosis, congestive heart failure, obesity, oral contraceptives, benzodiazepines, barbiturates and ethanol. J Pharm Sci, 1979 68 1358-1366... 

THC is the active component in marijuana, and ethanol is the alcohol in alcoholic beverages. Explain why drug screenings are able to detect the presence of THC but not ethanol weeks after these substances have been introduced into the body. 

This chapter addresses the toxic effects of the mixtures of two recreational toxins, ethanol and tobacco. These are the most abused chemicals in the world and both are used recreationally worldwide. There are many other recreational drugs in use. These include marijuana, cocaine, heroin, and methamphetamines for example. These other recreational drugs are not addressed here, except where they are components of xenobiotic mixtures that produce unanticipated toxic effects. At this time there is a substantial volume of information that describes unanticipated toxic effects of ethanol and tobacco when used in conjunction with other toxicants. That information is the subject of this chapter. 

Lukas SE, Orozco S. Ethanol increases plasma delta(9)-tetrahydrocannabinol (THC) levels and subjective effects after marijuana smoking in human volunteers. Drug Alcohol Depend 2001 64(2) 143 9. 

Little is known about the cause of ADHD and no cure has been found for it. It has been shown that maternal ingestion of lead or ethanol as well as smoking or marijuana use are causative for ADHD. Both tobacco and marijuana smoke are complex mixtures of thousands of individual chemicals and the chemical (s) responsible for inducing ADHD have not been identified. It has also been shown that exposures to mixtures of artificial colors and flavors are synergistically neurotoxic. 

Whitfield RM, Bechtel LM, Starich GH (1997) The impact of ethanol and Marinol/marijuana usage on HlV-t/AlDS patients undergoing azidothymidine, azidothymidine/dideoxy-cytidine, or dideoxyinosine therapy. Alcohol Clin Exp Res 21 122-127... 

Mason AP, McBay AJ (1984) Ethanol, marijuana, and other drug use in 600 drivers killed in single-vehicle crashers in North Carolina, 1978-1981. J Forensic Sci 29 987-1026... 

Table IV-1-10 summarizes the properties of drugs of abuse. These include the CNS stimulants (cocaine j and amphetamines), the CNS depressants (benzodiazepines, barbiturates, and ethanol), the opioids j (morphine, heroin, methadone, fentanyl, and others), the hallucinogens (marijuana and other j...

Answer A. The signs and symptoms are characteristic of a CNS stimulant that facilitates the activity of amines in both the CNS and the periphery. Amphetamines promote the release of NE from sympathetic nerve endings, causing CV stimulation and pupillary dilation. In the CNS, they enhance the actions of DA, NE, and 5HT, causing behavioral excitation and a psychotic state that may be difficult to distinguish from schizophrenia. Ethanol, marijuana, fentanyl, and flunitrazepam (a benzodiazepine that has been used in date rape ) are all CNS depressants. 

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