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Lungs, ischemia-reperfusion injury

Marts BC, Baudendistel LJ, Naunheim KS, Dahms TE (1993) Protective effect of 2-chloroadenosine on lung ischemia reperfusion injury. J Surg Res 54(6) 523-529 Matot I, Jurim O (2001) Protective effect of acadesine on ischemia-reperfusion lung injury. Anesth Analg 92(3) 590-595... [Pg.229]

Naidu BV, Krishnadasan B, Farivar AS, Woolley SM, Thomas R, Van Rooijen N, Verrier ED, Mulligan MS (2003) Early activation of the alveolar macrophage is critical to the development of lung ischemia-reperfusion injury. 1 Thorac Cardiovasc Surg 126(l) 200-207... [Pg.230]

Belperio JA, Keane MP, Burdick MD, et al. CXCR2/CXCR2 ligand biology during lung transplant ischemia-reperfusion injury. J Immunol 2005 175 6931-6939. [Pg.151]

Maxey TS, Enelow RI, Gaston B, Kron IL, Laubach VE, Doctor A (2004) Tumor necrosis factor-alpha from resident lung cells is a key initiating factor in pulmonary ischemia-reperfusion injury. J Thorac Cardiovasc Surg 127(2) 541—547 Mayer AM, Pittner RA, Lipscomb GE, Spitzer JA (1993) Effect of in vivo TNF administration on superoxide production and PKC activity of rat alveolar macrophages. Am J Physiol Lung Cell Mol Physiol 264(1 Pt 1) L43-L52... [Pg.229]

Neely CF, Keith IM (1995) A1 adenosine receptor antagonists block ischemia reperfusion injury of the lung. Am 1 Physiol 268(6 Pt 1) L1036-L1046... [Pg.230]

Gaseous nitric oxide is a short-lived molecule that has been used in the treatment of patients with primary pulmonary hypertension and is used in subgroups of severely ill and hypoxic children with persistent pulmonary hypertension of the newborn, in preterm infants of less than 34 weeks gestation, and in adults with acute lung injury and adult respiratory distress syndrome. There are some reports of its use for intestinal ischemia, reperfusion injury, thrombotic disorders, and sickle cell crises. [Pg.2538]

SOD3 knockout mice are apparently healthy but are more sensitive to hyperoxic lung injury [168], to ischemia-reperfusion injury [169], and to the diabetogenic action of alloxan [170]. [Pg.127]

The hallmark of an inflammatory response in the lung is the presence of infiltrating leukocytes. This process can occur in the context of a variety of disorders, including trauma, infection, autoimmune diseases, idiopathic interstitial pneumonias, asthma, chronic bronchitis, acute respiratory distress syndrome (ARDS), exposure to environmental/occupational noxious agents, cancer, aUograft rejection, and ischemia-reperfusion injury. The course of inflammation in these disease states is defined by the delicate balance and nature of inflammatory mediators expressed in the context of lung inflammation, and the specific leukocyte populations recmited in response to lung injury. [Pg.413]

Suda T, Mora BN, D Ovidio F, Cooper JA, Hiratsuka M, Zhang W, Mohanakumar T, Patterson GA. In vivo adenoviius-medialed endothelial nitrie oxide synthase gene transfer ameliorates lung allograft ischemia-reperfusion injury. J Thorac Cardiovasc Surg 2000 119 297 304. [Pg.472]

Daddi N, Suda T, Kozower BD, Kanaan S, Mohanakumar TS, Patterson GA. Recipient intramuscular cotransfection of naked plasmid TGF betal and IL-10 ameliorates lung graft ischemia-reperfusion injury. American Association for Thoracic Surgery, San Diego, May 9,2001. [Pg.473]

Adenosine A3 Receptor and Ischemia-Reperfusion-Induced Lung Injury... [Pg.215]

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See also in sourсe #XX -- [ Pg.143 ]



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Ischemia reperfusion

Lung injury

Reperfusion

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