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Hypoglycemia caused

Oral anticoagulants also may potentiate hypoglycemia caused by oral hypoglycemic agents, and may enhance phenytoin toxicity. [Pg.261]

Severe hypoglycemia caused by hyperinsulinism may occur in cases of insulin overdose, when food intake is decreased or delayed, during periods of excessive exercise, or in patients with brittle diabetes. [Pg.628]

Severe hypoglycemia caused by hyperinsulinism may occur with insulin overdose, decrease or delay of food intake, or excessive exercise and in those with brittle diabetes. [Pg.630]

In healthy volunteers hypoglycemia caused significant deterioration in short-term attention, whereas sustained attention and intelligence scores did not deteriorate (79). [Pg.397]

Janda A, Salem C. Hypoglykamie durch Lidocain-Uberdosierung. [Hypoglycemia caused by lidocaine overdosage.] Reg Anaesth 1986 9(3) 88—90. [Pg.675]

Hasegawa Y, Formato JE, Latour LL, Gutierrez JA, Liu KF, Garcia JH, Sotak CH, Fisher M (1996) Severe transient hypoglycemia causes reversible change in the apparent diffusion coefficient of water. Stroke 27 1648-1655 discussion 1655-1646... [Pg.129]

Oral diazoxide (Proglycem) increases serum glucose level by inhibiting insulin release from the beta cells and stimulating release of epinephrine (Adrenalin) from the adrenal medulla. Oral diazoxide is used to treat chronic hypoglycemia caused by hyperinsulinism resulting from islet cell cancer or hyperplasia. It is not for hypoglycemic reactions. [Pg.340]

Insulin-dependent diabetic patients with advancing renal failure often develop more frequent episodes of hypoglycemia resulting from decreased renal degradation of insulin. Emaciated nondiabetic patients may also develop spontaneous hypoglycemia caused by low glycogen stores and the decreased contribution by the diseased kidneys to gluconeogenesis (Bll, W5). [Pg.94]

As described, the secretion of epinephrine in response to stress, trauma, extreme exercise, or hypoglycemia causes a rapid mobilization of energy stores, that is, glucose from the liver and fatty acids from adipose tissue. The reaction in which NE is methylated to form E is mediated by the enzyme phenylethanolamine-N-methyltransferase (PNMT). Although the enzyme occurs predominantly in the chromaffin cells of the adrenal medulla, it is also... [Pg.484]

Diazoxide is given as intravenous boluses and has a duration of action of several hours. Diazoxide opens potassium channels, thus hyperpolarizing and relaxing smooth muscle cells. This drug also reduces insulin release and can be used to treat hypoglycemia caused by insulin-producing tumors. The toxicity of diazoxide includes hypotension, hyperglycemia, and salt and water retention. [Pg.103]

Diazoxide is administered orally in the management of hypoglycemia caused by hyperinsulinism associated with inoperable islet cell adenoma or carcinoma or extrapancreatic malignancy in adults. [Pg.1164]

Exogenously administered animal insulin can be distinguished from endogenous insulin (ie, in a patient with hypoglycemia caused by insulinoma) by determination of C peptide (present with endogenous insulin... [Pg.95]


See other pages where Hypoglycemia caused is mentioned: [Pg.153]    [Pg.267]    [Pg.276]    [Pg.312]    [Pg.339]    [Pg.1787]    [Pg.45]    [Pg.396]    [Pg.444]    [Pg.454]    [Pg.665]    [Pg.86]    [Pg.742]    [Pg.1765]    [Pg.3232]    [Pg.3240]    [Pg.123]    [Pg.866]    [Pg.890]    [Pg.763]    [Pg.308]    [Pg.874]    [Pg.576]    [Pg.853]    [Pg.165]    [Pg.420]    [Pg.35]   
See also in sourсe #XX -- [ Pg.35 , Pg.95 , Pg.96 , Pg.132 , Pg.191 , Pg.351 , Pg.455 ]




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