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Hypoglycemia insulin excess causing

Hypoglycemia Hypoglycemia may result from excessive insulin dose or may be caused by Increased work or exercise without eating food not being absorbed in the usual manner because of postponement or omission of a meal or in illness with vomiting, fever, or diarrhea when insulin requirements decline. [Pg.298]

Severe hypoglycemia caused by hyperinsulinism may occur in cases of insulin overdose, when food intake is decreased or delayed, during periods of excessive exercise, or in patients with brittle diabetes. [Pg.628]

Severe hypoglycemia caused by hyperinsulinism may occur with insulin overdose, decrease or delay of food intake, or excessive exercise and in those with brittle diabetes. [Pg.630]

Co-trimoxazole can cause reversible hypoglycemia, which may be prolonged, particularly in patients with risk factors for hypoglycemia. Common risk factors include compromised renal function, prolonged fasting, malnutrition, and the use of excessive doses. It has been postulated that the sulfonamide mimics the action of sul-fonylureas, stimulating pancreatic islet cells to secrete insulin. In elderly people, co-trimoxazole-induced hypoglycemia can cause altered mental state (1146,1147). [Pg.653]

Hypoglycemia is a primary complication of insulin therapy and may result from either an excess of insulin or a lack of glucose, or both. Severe hypoglycemia may cause headache, confusion, double vision, drowsiness, and convulsions. The treatment of this hypoglycemia may include the administration of glucose or glucagon. [Pg.504]

Because insulin stimulates the uptake of glucose into tissues where it may be immediately oxidized or stored for later oxidation, this regulatory hormone lowers blood glucose levels. Therefore, one of the possible causes of Bea Selmass s hypoglycemia is an insulinoma, a tumor that produces excessive insulin. [Pg.483]

The exact cause of reactive hypoglycemia is difficult to determine, but most researchers believe that the main cause is a shortage of glucagon, resulting in an inability to raise blood glucose levels when necessary. Others speculate that the cause is a hypersensitive response to the release of the normal physiological hormone called epinephrine, a reaction that is the opposite of epinephrine s normal effect on blood sugar, which is to raise it. Still others consider excessive insulin production to be the culprit. [Pg.435]


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See also in sourсe #XX -- [ Pg.162 ]




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