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Humans plasma glucose concentrations

First-line therapy includes nutritional and exercise interventions for all women, and caloric restrictions for obese women. If nutritional intervention fails to achieve fasting plasma glucose levels less than or equal to 105 mg/dL, 1-hour post-prandial plasma glucose concentrations less than or equal to 155 mg/dL, or 2-hour postprandial levels less than or equal to 130 mg/dL, then therapy with recombinant human insulin should be instituted glyburide may be considered after 11 weeks of gestation. [Pg.368]

Cellular Glucose Concentration The concentration of glucose in human blood plasma is maintained at about 5 mM. The concentration of free glucose inside a myocyte is much lower. Why is the concentration so low in the cell What... [Pg.599]

Wolf R, Rosche F, Hoffmann T, et ah Im-munoprecipitation and liquid chromatographic-mass spectrometric determination of the peptide glucose-dependent insuli-notropic polypeptides GIP1-42 and GIP3-42 from human plasma samples. New sensitive method to analyze physiological concentrations of peptide hormones. /. Chromatogr. A (2001) 926 21-27. [Pg.178]

Fig. 23. Relationship between glucose concentrations of human blood plasma samples measured by a glucose-sensitive FET sensor and by a conventional enzymatic method (F = 1.007X 14, r = 0.988, n = 101). (Re-... Fig. 23. Relationship between glucose concentrations of human blood plasma samples measured by a glucose-sensitive FET sensor and by a conventional enzymatic method (F = 1.007X 14, r = 0.988, n = 101). (Re-...
FIGURE 4.4 Changes in plasma metabolite and hormone levels with exercise. The human subjects were at n st at time zero. Exercise was coitiinucd for 250 min, and concentrations of plasma glucose (O), glucagon (A), and insulin ) were measured at the indicated times. (Redrawn with permission from Felig and Wahren, J975.)... [Pg.165]

The clinical manifestations and treatment of hypophosphatemia in the horse have not been reported and in humans there is no good evidence for commencing treatment in the absence of clinical signs (Bugg Jones 1998). Treatment options reported in small animals include i.v. (0.01-0.03 mmol/h per kg) and oral (0.5-2 mmol/kg daily) potassium phosphate or sodium potassium phosphate (Macintire 1997). The author has used this dose rate of phosphate in mature horses, with apparent clinical success. The potential effects of potassium phosphate on the plasma potassium concentration must be considered before commencing treatment. Glucose 1-phosphate (Bollaert et al 1995) and sodium phosphate administration i.v. have also been reported in humans. The safety of these treatments has not been evaluated in the horse. [Pg.356]


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See also in sourсe #XX -- [ Pg.361 ]




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