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Genetic risks

The majority of people who smoke never develop lung cancer. Genetic risk factors may predispose certain smokers to lung cancer. After adjustments for age, smoke exposure, occupation, and gender, relatives of a lung cancer patient have approximately a twofold risk of developing lung cancer. The... [Pg.1324]

Moatti D, Faure S, Fumeron F, et al. Polymorphism in the fractalkine receptor CX3CR1 as a genetic risk factor for coronary artery disease. Blood 2001 97(7) 1925-1928. [Pg.227]

Ghilardi G, Biondi ML, Turri O, Guagnellini E, Scorza R. Internal carotid artery occlusive disease and polymorphisms of fractalkine receptor CX3CR1 a genetic risk factor. Stroke 2004 35(6) 1276-1279. [Pg.227]

Marteau TM, Lerman C. Genetic risk and behavioural change. BMJ 2001 322 1056-1059. [Pg.10]

Frosst P, Blom HJ, Milos R et al. A candidate genetic risk factor for vascular disease a common mutation in methylene-tetrahydrofolate reductase. Nature Genet 1995 10 111-113. [Pg.308]

Sleegers, K., Roks, G., Theuns, J. etal. Familial clustering and genetic risk for dementia in a genetically isolated Dutch population. Brain 127 1641-1649, 2004. [Pg.665]

Sankaranarayanan, K. 1991a. Ionizing radiation and genetic risks II. Nature of radiation-induced mutations in experimental mammalian in vivo systems. Mutat. Res. 258 51-73. [Pg.1749]

Cigarette smoking is responsible for about 80% of lung cancer cases. Other risk factors include exposure to respiratory carcinogens (e.g., asbestos, benzene), genetic risk factors, and history of other lung diseases (e.g., tuberculosis, pulmonary fibrosis). [Pg.712]

These results suggest that the taxon is overinclusive It includes 28% of low-risk participants—instead of the 10% predicted by Meehl s theory—and misses some cases that later become symptomatic. This might mean that the identified taxon is not isomorphic with specific genetic liability for schizophrenia and reflects a construct that is overlapping, but not identical to, the genetic risk factor. Another explanation is that the DSM criteria for schizophrenia and spectrum conditions may be too broad. Tyrka et al. (1995) proposed this hypothesis and estimated that at least two-thirds of the misses (symptomatic cases not assigned to the taxon) can be accounted for by errors in the taxon classification scheme, but the remaining misses are due to... [Pg.119]

Sorbi, S., et al., "Genetic Risk Factors in Familial Alzheimer s Disease," Mech Ageing Dev., 122, 1951-1960 (2001). [Pg.57]

The prototypical form of error in the health care system that could be reduced by a systems approach is medication error. The kind of error identified in the literature—overdose of chemotherapy, injection of the wrong drug, etc.—sometimes leads to either injury or death, the kinds of harm that are the central concern of after-the-fact medical liability adjudication. Phar-macogenomics introduces not only another conception of harm—genetic risks—but also new ways of developing and prescribing drugs. [Pg.189]

The role of Lp(a) as a primary genetic risk factor for coronary heart disease, therefore, seems firmly established. However, in blacks, despite mean Lp(a) levels twice as high as in whites, the incidence of cardiovascular disease is apparently identical (G37, S40). The role of Lp(a) in atherosclerotic vascular disease in blacks therefore remains to be established. [Pg.93]

A genetic consultation is a health service that provides information and support to people who have, or may be at risk for, genetic disorders. During a consultation, a genetics professional meets with an individual or family to discuss genetic risks or to diagnose, confirm, or rule out a genetic condition. [Pg.36]

Djalali-Behzad G, Hussain S, Osterman-Golkar S, et al. 1981. Estimation of genetic risks of alkylating agents. VI. Exposure of mice and bacteria to methyl bromide. Mutat Res 84 1-9. [Pg.95]


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See also in sourсe #XX -- [ Pg.61 ]




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