Ethanol for poisoning with

What are some of the alternatives for this procedure Well, one can use NaOH (lye) in place of KOH but the yields will go down. Also, what about using denatured alcohol instead of pure ethanol Denatured alcohol is ethanol contaminated with 5-10% methanol. The methanol is there because it is poisonous and prevents people from using the ethanol for drinking. This means that places like the giant hardware stores can carry gallons of cheap contaminated ethanol as many of you have discovered. This product CAN be used. 

DETAILS - Colchicine is a plant drug used in the treatment of gout. It is a very efficient poison in view of its very low toxic dose and the fact that an autopsy will show only the symptoms of acute gastroenteritis. This does not, however, mean it is undetectable. It only means that it is likely to be overlooked. It is also very useful as a dart poison, especially when nicotine is used as a binder. Colchicine is great for small caliber (,22)bullets, as they usually won t hold enough of most other poisons to do any good. It dissolves slowly in water, but faster in dilute ethanol (liquor). As with any plant alkaloid, it is best to harvest the... 

Contraindications Intestinal obstruction, GI tract not anatomically intact patients at risk of hemorrhage or GI perforation, if use would increase risk and severity of aspiration not effective for cyanide, mineral acids, caustic alkalis, organic solvents, iron, ethanol, methanol poisoning, lithium do not use charcoal with sorbitol in patients with fructose intolerance, hypersensitivity to charcoal or any component of the formulation... 

Third, for forensic cases in which there is a question of poisoning with ethanol, a sample of blood could be analyzed for FAEE quantitation to assess prior ethanol administration. It is not yet known if there are any causes for a false-positive FAEE test or a false-negative FAEE value. It is possible that ingestion of certain medications or foodstuffs will make the FAEE test falsely positive, and it is also possible that interferences exist that block the detection of FAEE. No causes of false-positive or falsenegative tests have been yet identified, but there is always a possibility that one will appear as more research is done in the field. In addition, one cannot use the level of blood FAEE 24 h after drinking alcohol to predict the peak blood ethanol concentration much earlier. 

The one-carbon alcohol methanol also is metabohzed by ADH and ALDH, with damaging consequences (see below). Competition between methanol and ethanol for ADH forms the basis of the use of ethanol in methanol poisoning. Several drugs inhibit alcohol metabolism, including... 

Fig. 3. Insensitivity of the rate for ethanol and ethyl ether dehydration (O) on catalyst poisoning with Na ions ( and O)-...

Eirst aid treatment for humans accidentally poisoned with 1080 includes irmnediate emesis and gastric lavage followed by an oral dose of magnesium sulfate or sodium sulfate to remove the poison from the alimentary tract before absorption of lethal quantities can occur. When the stomach is emptied, oral administration of ethanol may be beneficial. The patient should be put at complete rest and given barbiturates having moderate duration of action, such as sodium amytol, to... 

Potassium cyanide [15]-50-8 M 65.1, m 634 , d 1.52. A saturated solution in H20-ethanol (1 3) at 60° was filtered and cooled to room temperature. Absolute EtOH was added, with stirring, until crystallisation ceased. The solution was again allowed to cool to room temperature (during 2-3h) then the crystals were filtered off, washed with absolute EtOH, and dried, first at 70-80° for 2-3h, then at 105° for 2h [Brown, Adisesh and Taylor J Phys Chem 66 2426 7962]. Also purified by vacuum melting and zone refining. HIGHLY POISONOUS. 

Attempts to diminish the overall metabolism of trichloroethylene might be useful (e.g., hypothermia, mixed-function oxidase inhibitors, competitive inhibitors of trichloroethylene metabolism [i.e., P-450 substrates]), if instituted soon enough after trichloroethylene exposure. Catecholamines (especially beta agonists) act in concert with trichloroethylene, increasing the risk of cardiac arrhythmias. Hence, catecholamines should be administered to patients only in the lowest efficacious doses and for certain limited presentations of trichloroethylene poisoning. Ethanol should also be avoided because concurrent exposure to trichloroethylene and ethanol can cause vasodilation and malaise and may potentiate central nervous system depression at high dosage levels of either compound. 

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