Veins, structure

G.E., abbrev. (Gewichtseinheit) unit of weight. Geilder, n. veins, veined structure system of blood vessels. 

Urashima, Y. (1953) The vein structures of the Gogo-Myaku (No. 5 vein) and the occurrence of iron sluphide minerals at the Konomai mine, Hokkaido. Mining Geology, 3, 174-180 (in Japanese with English abst.). 

Perlite is an expanded volcanic glass. It is thin and diaphanous except where interconnections of glassy bubbles give the impression of veins. If the veins are separated from the glassy film, they may loosely resemble chrysotile asbestos. The separation is seldom complete. Close observation will detect the film fragments associated with the vein structure. 

Fin. 7. Tensile fracture surfaces showing the typical vein structure in amorphous metals (a) CusnZrso (b) Co7(iFe5.Sii5B ii. 

Graphitic conductors, pyrite concentrations, buried channels, fault structures Buried channels, fault, vein structures Sulphide minerals, clay mineralogy changes Geologic structures, regional and local facies changes buried channels, local unconformity mapping... 

A total of 49% of the lesions showed a relationship to the liver capsule, 7% presented a relationship to the central portal vein structures and only 29% of the metastases were at a location which was classified as easy. 

Fig. 25.7. The graph shows the distribution ofthe liver metastases with respect to the localization of the lesion. A localization was classified as easy if the lesion was sufficiently surrounded by normal liver parenchyma without relationship to any of the other listed structures. A lesion was classified as paracaval if there was a contact to the vena cava inferior. Other important relationships were the liver capsule, the gall bladder, the bowel and the central portal vein structures (including the central bile ducts). A lesion was classified as subcardial, if the lesion was located in liver segment 11 and the distance between the lesion and the pericardium was less than 8 mm...

FIGURE 5.38 Pictorial presentation of the microscopic structure of the liver. The picture shows the classical liver lobulus. The functional acinus and its three zones are at the left. The acinal zones are marked by numbering them 1-3. These zones correspond to the direction of blood flow from the portal arteries (PA) to the terminal veins (TV). Zone I corresponds to the periportal area in classical liver pathology, zone 2, the interlobular region (midzone), and zone 3, centrelobular region. ... 

It was therefore of some interest to so modify the molecule as to maximize this particular activity at the expense of the side effects. In much the same vein as the work on cocaine, the structural requirements for the desired activity had at one time been whittled down to embrace in essence an a-substituted phenylacetic acid ester of ethanolamine (S3). 

Streifen, m. band, strip, stripe, stria streak, vein (in marble, etc.) strap, -gefiige, n. banded structure, -kohle, /. banded coal, -spektrum, n. band spectrum. 

Vessel identity represents one of the major differentiation processes during blood vessel formation. Arteries and veins are structurally and functionally... 

The veins are composed mostly of quartz and a small amount of sulfide minerals (pyrite, pyrrhotite, arsenopyrite, chalcopyrite, sphalerite, and galena), carbonate minerals (calcite, dolomite) and gold, and include breccias of the host rocks with carbonaceous matters. Layering by carbonaceous matters has been occasionally observed in the veins. Banding structure, wall rock alteration and an evidence of boiling of fluids that are commonly observed in epithermal veins have not been usually found. 

Takahashi, M. Ishiyama, T. and Mizuta, T. (1998) Structure and environment of formation of the Hosen No. 5 and Ryosen No. 5 gold-quartz veins, Hishikari mine. Japan. Rep. Inst. Appl. Earth Sci. Dep. Geosci. Akita U., 63, 55-72 (in Japanese with English abst.). 

Watanabe, Y. (1991) Mineralization ages of Ofukesi, Shizukari, Yakumo and Jokoku deposits and structural movements related to vein-type mineralization in southwest Hokkaido. Mining Geology, 41, 141-146. 

Portal hypertension is a consequence of increased resistance to blood flow through the portal vein. Increased resistance is usually due to restructuring of intrahepatic tissue (sinusoidal damage) but may also be caused by presinusoidal damage such as portal vein occlusion from trauma, malignancy, or thrombosis. A third (and the least common) mechanism is outflow obstruction of the hepatic vein. This latter damage is posthepatic, and normal liver structure is maintained. This chapter will focus on portal hypertension caused by intrahepatic damage from cirrhosis. 

Thus, important structure/property relationships are emerging that are relevant to electronic and optical materials applications for these materials. In a different vein, side chain crystallization has resulted in the first liquid crystalline inorganic and organometallic macromolecules, viz., unusual poly(dialkoxy-phosphazenes) described by Allcock (p. 250) and Singler (p. 268). 

The adenohypophysis does not have a direct anatomical connection with the hypothalamus therefore, regulation of hormone secretion by way of neuronal signals is not possible. Instead, these two structures are associated by a specialized circulatory system and the secretion of hormones from the adenohypophysis is regulated by hormonal signals from the hypothalamus (see Figure 10.2). Systemic arterial blood is directed first to the hypothalamus. The exchange of materials between the blood and the interstitial fluid of the hypothalamus takes place at the primary capillary plexus. The blood then flows to the adenohypophysis through the hypothalamic-hypophyseal portal veins. Portal veins are blood vessels that connect two capillary beds. The second capillary bed in this system is the secondary capillary plexus located in the adenohypophysis. 

---