Thyroid releasing hormone

Reports of the effects of Li+ upon the thyroid gland and its associated hormones are the most abundant of those concerned with the endocrine system. Li+ inhibits thyroid hormone release, leading to reduced levels of circulating hormone, in both psychiatric patients and healthy controls [178]. In consequence of this, a negative feedback mechanism increases the production of pituitary TSH. Li+ also causes an increase in hypothalamic thyroid-releasing hormone (TRH), probably by inhibiting its re-... 

Thyroid-Releasing Hormone Toxic Substances Control Act Thyroid Stimulating Hormone Treatment Technique Threshold of Toxicological Concern Target-organ Toxicity dose Unscheduled DNA Synthesis Uncertainty Factor United Nations... 

Angiotensin II, administered into the central nervous system, increases the release of luteinizing hormone, adrenocortical hormone, thyroid-releasing hormone, (3-endorphin, vasopressin, and oxytocin from the anterior pituitary. In contrast, centrally administered angiotensin II inhibits the release of anterior pituitary growth hormone and prolactin. 

Challenge tests Releasing hormone challenge tests to test pituitary responsiveness to hypothalamic hormones (GnRH, corticotropin releasing hormone, thyroid releasing hormone)... 

Less common etiologies include central nervous system lesions that physically compress the pituitary stalk and interrupt tonic hypothalamic dopamine secretion, resulting in hyperprolactinemia. Increased thyroid-releasing hormone (TRH) concentrations in hypothyroidism can stimulate prolactin secretion and cause hyperprolactinemia. During conditions of renal or liver compromise, the... 

Of course there are other chemicals in the body s Action/Reaction Factor closet that positively regulate prolactin. The major ones are GnRH, TRH (thyroid Releasing Hormone) and VIP (Vasoactive Intestinal Polypeptide). By the way, hyper-stimulation of the nipples may have a stimulatory effect upon prolactin release as well. But that is one we will leave alone. 

TRH Thyroid-releasing hormone TSH Thyroid-stimulating hormone... 

The thyroid releasing hormone binds to receptors in the pituitary and like many other hormones activates adenyl cyclase and increases the intracellular levels of cyclic AMP. 

The discovery of the existence and the determination of the structure of the thyroid-releasing hormone have led to important clinical applications with respect to the diagnosis of thyroid diseases. Only a few examples will be presented. 

Hormones secreted in the hypothalamus, which releases pituitary hormones, are believed to bind to specific receptors of the membranes of the pituicyte. This action is dependent upon the presence of Ca Therefore, it has been proposed that the hormone depolarizes the cell membrane increasing the permeability to calcium which would, in turn, release the hormone. Whatever the role of calcium, it is known in the case of thyroid releasing hormone that it stimulates adenylcyclase and cAMP. 

Hypothalamus Oxytocin, vasopressin, thyroid releasing hormone, LH-RH, somatostatin... 

The linking of one molecule of MIT with one of DU results in the formation of T3, whilst two molecules of DIT combine in the formation of T4. T4 can also be converted into T3 by the 5 -deiodinase enzyme system. Within the body, the control system associated with the production of these hormones is directed by TSH (thyroid stimulating hormone) and TRH (thyroid releasing hormone). 

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