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String defects

These string-like defects are considered to be a kind of zigzag defect. In each string defect, helical patterns can be observed. Moreover, in each adjacent string, the directions of the helical patterns are opposite. [Pg.229]

Step movement during chemisorption appears to be a general phenomenon. Real-time images observed (Figure 4.4) for chlorine chemisorption at Cu(110) indicate that nucleation takes place at a defect site, resulting in a single string... [Pg.53]

Chia Yes, we can look at Insc localization in G2. With String, all the cells are in G2, so you can just look at the embryo. In some ways it is a more convincing result. The defects that we see are seen during mitosis. [Pg.152]

Fibrillin microfibrils are widely distributed extracellular matrix assemblies that endow elastic and non elastic connective tissues with long-range elasticity. They direct tropoelastin deposition during elastic fibrillogenesis and form an outer mantle for mature elastic fibers. Microfibril arrays are also abundant in dynamic tissues that do not express elastin, such as the ciliary zonules of the eye. Mutations in fibrillin-1—the principal structural component of microfibrils—cause Marfan syndrome, a heritable disease with severe aortic, ocular, and skeletal defects. Isolated fibrillin-rich microfibrils have a complex 56 nm beads-on-a-string appearance the molecular basis of their assembly and... [Pg.405]

In semi-automatic moulding the operator may inspect each unit for features including colour, dispersion and uniformity of colour, standard and uniformity of gloss or mattness, and the presence or otherwise of defects like silver streaks or stringing. However, it is preferable if assessments can be made in numerical terms, expressed in units that may be compared quantitatively and subjected easily to statistical treatment and analysis. It is often convenient in the course of... [Pg.154]

Figure 21. Model of dislocation string detachment from point defect. Pinning in A and B points supposed rigid /i, /j are lengths of vibrating dislocation segments. Arrow shows direction of dislocation slipping and outer stress (p is critical detachment angle. Figure 21. Model of dislocation string detachment from point defect. Pinning in A and B points supposed rigid /i, /j are lengths of vibrating dislocation segments. Arrow shows direction of dislocation slipping and outer stress (p is critical detachment angle.
In panel (d) a strikingly different type of defect structure is encountered, the so-called tears. It should be noted that these correspond to much larger defects (compare the scan size). In the work of Russell and coworkers it was found that three types of failure were present in the rubbed polyimide films, namely scratches, tears, and strings of islands, all of which are aligned along the mbbing direction. Local overshearing of the film was held responsible for the observed formation of... [Pg.180]

Casanova et al. (2002) have compared the brains from autistic individuals with normal brains and found that in Nissl stained vertical sections taken from Brodmann s areas 9, 21, and 22, the minicolumns in the normal and autistic brains have a somewhat different structure. In the autistic brains, the minicolumns are smaller in width, and the neurons within the vertical strings are less tightly packed, leading the authors to conclude that per unit volume of cerebral cortex, the minicolumns are more numerous in the cortices of autistic individuals. Since studies by earlier authors have shown that there is no abnormality in cell density in the cortices of autistic individuals compared to controls (Coleman et al., 1985 Bailey et al., 1993), Casanova et al. (2002) suggest that autism is the consequence of a defect in migration of cells into the cortex during development. [Pg.63]


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See also in sourсe #XX -- [ Pg.230 ]




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