Promotion of elongation

In the reverse direction, protonation of the phosphate of glucose-1-phosphate destabilizes the glycosidic bond and promotes formation of a glucosyl oxocarbonium ion-phosphate anion pair. In the subsequent step, the phosphate anion becomes essential for promotion of the nucleophilie attack of a terminal glucosyl residue on the carbonium ion. This sequence of reactions brings about a-1,4-glycosidic bond formation and primer elongation. 

Both compounds are rapidly translocated herbicides that selectively kill broad-leaved weeds. They mimic the natural auxin, indol-3-ylacetic acid, which is responsible for promoting cell elongation and hence cause unrestrained growth. This uses up all the available nutrients, leading to the death of the plant. A review of the pyridinecarboxylic acids has been published (B-75MI10702). 

Salt. When compared on a molar basis, the mineral ion salts (ammonium, calcium, rubidium, copper, silver, lead, manganese, cobalt, potassium, and sodium, and cyclohexylamine salt) were as effective as the free gibberellic acid in promoting stem elongation (10,14). As shown in Figure 1, the potassium and zinc salts of A3 were as active as the acid in promoting the growth of d-1 dwarf maize. 

Radiclonic acid and its 0-acetyl and 12-hydroxy derivatives were isolated from the culture filtrate of a PenciIlium sp. fungus. These compounds promoted root elongation in many types of seedlings, the maximum activity was about 25 ppm for Chinese cabbage seedlings for rice seedlings, it was inhibitory above 100 ppm and stimulatory below this concentration (15, 16). 

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