Prepatent period

IL-12 and IFN-y-deficient mice fail to show any unusual susceptibility to schistosomiasis (La Flamme et al., 2001 b Patton et al., 2001), findings that suggest that the Th1 response that develops during the prepatent period of infection serves no essential host-beneficial function. However, as mentioned above, IL-12/IL-10—/— mice develop a severe form of schistosomiasis that is related to the development of excessively strong and polarized Th2 responses (Hoffmann et al., 2000). 

Prepatent period of Taenia ovis in dogs (correspondence). New Zealand Veterinary Journal, 28 193-4. 

Equids are highly susceptible to T. brucei infection, with a very high mortality rate within 14-90 days in imtreated horses. The prepatent period, the period from infection to parasitemia, is approximately 6-10 days. Locomotor ataxia is the earliest clinical sign. Remittent high fever (41.7°C/107°F) occurs between intervening periods when the... 

Disease caused by T. congolense is rare in horses. The prepatent period is 17 days and the parasitemia is imimpressive. Edematous plaques become evident after about 10 days, and may recur during the infection. Generalized edema becomes evident from about 14 days postinfection. Keratitis may develop. T. congolense infection is relatively mild in horses normal appetite despite a moderate anemia and clinical signs that resolve spontaneously in about 6 veeks. 

Equine disease caused by T. vivax is usually, but not always, relatively iimocuous. The prepatent period is about 16 days. Parasitemia is cyclic and is accompanied by fever. Between these febrile episodes, urticarial plaques may become evident in the skin and dependent edema develops. In more severe cases, ataxia may develop in the first few days after infection, with muscle tremors occurring around 2 weeks after infection. The ataxia and anorexia are progressive. Keratitis with associated comeal opacity may develop. 

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