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Pathogens - Anti-Animal

Pathogens may be introduced via inhalation, contact, ingestion, injection, or vector. [Pg.127]

Varies depending on the pathogen, but is generally on the order of days to weeks. Exposures to extremely high doses of some pathogens may reduce the incubation period to as short as several hours. [Pg.127]

Emergency Action for Chemical and Biological Warfare Agents [Pg.128]

Although pathogens are selected to target a specific animal species, there is the possibility that the disease may migrate to humans. [Pg.128]

Immediately isolate an area around any liquid or solid contamination for at least 100 feet in all directions. Adjust the initial isolation distance as appropriate. Based on the type of release, amount of material aerosolized and local conditions (e.g., weather, population density, time of day), shelter-in-place until the initial cloud passes may be the most appropriate course of action since timely evacuation of the threatened downwind population may not be possible. [Pg.128]


Use male Wistar or Sprague-Dowley rats. Results are more reproducible in young (200-250 g) e.g., animals. It is important to use pathogen-free animals, since pulmonary disorders (pneumonia) markedly affect the pulmonary uptake of radiolabeled anti-ACE MAb 9B9 (10). Devote at least four animals in each experimental group (see Note 4). [Pg.246]

Agroterrorism, which may involve anti-plant pests, in addition to anti-plant and anti-animal pathogens. [Pg.1529]

However, in case of acute clinical mastitis, it is widely accepted that animal welfare considerations should take prevalence. If both farmer and veterinarian are not familiar with non-antibiotic treatments, they should be advised to use broad-spectrum antibiotics immediately, because any delay (e.g. the 2-3 days it often takes between diagnosis and the return of microbiological test results) may seriously harm the animal. This approach should, however, only be taken after a sound clinical diagnosis, since antibiotic treatments themselves may lead to dramatic aggravation of the condition. For example, E. coli inflammations are able to develop into severe toxaemia, because increased levels of toxins are released into the animal tissues when E. coli cells are killed or stressed by antibiotic treatments. Also, if yeasts are the main cause or form part of the pathogen complex that causes mastitis, their growth and proliferation may be supported by the administration of anti-bacterial antibiotics (Crawshaw et al., 2005). [Pg.206]

Wild animals get infected with parasites and pathogens, and while they may not always show external signs or symptoms, they can treat themselves by modifying their diets to a certain extent and benefit from their anti-parasitic qualities (Huffman, 2003 Huffman, 2006). [Pg.192]


See other pages where Pathogens - Anti-Animal is mentioned: [Pg.127]    [Pg.14]    [Pg.365]    [Pg.13]    [Pg.364]    [Pg.127]    [Pg.14]    [Pg.365]    [Pg.13]    [Pg.364]    [Pg.127]    [Pg.365]    [Pg.1609]    [Pg.364]    [Pg.289]    [Pg.222]    [Pg.148]    [Pg.728]    [Pg.409]    [Pg.403]    [Pg.261]    [Pg.56]    [Pg.208]    [Pg.77]    [Pg.86]    [Pg.292]    [Pg.131]    [Pg.139]    [Pg.198]    [Pg.8]    [Pg.458]    [Pg.282]    [Pg.18]    [Pg.2]    [Pg.193]    [Pg.16]    [Pg.230]    [Pg.260]    [Pg.521]    [Pg.302]    [Pg.281]    [Pg.172]    [Pg.99]    [Pg.9]    [Pg.960]    [Pg.448]    [Pg.117]    [Pg.120]    [Pg.521]    [Pg.352]    [Pg.356]   


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