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Lower extremities ankle

Instruct the patient regarding nonpharmacologic strategies including elevation of the affected extremity and anti-embolic exercises such as flexion/extension of the ankle (for lower extremity VTE) or hand squeezing/relaxation (for upper extremity VTE). [Pg.158]

Exts Right lower extremity with erythema and edema from the ankle to just below the knee. Warm to the touch. LLE within normal limits. [Pg.1080]

Miller, M.S. Use of topical recombinant human platelet-derived growth factor-BB (becaplermin) in healing of chronic mixed arteriovenous lower extremity diabetic ulcers. J. Foot Ankle Surg. 1999, 38 (3), 227-231. [Pg.301]

Yao ST, Hobbs JT, Irvine WT. Ankle systolic pressure measurements in arterial disease affecting the lower extremities. Br J Surg 1969 56 676-679. [Pg.459]

Baumgartner 1998 Critical lower extremity ischemia Critical ischemia, resistant to maximal medical therapy, not surgical candidates Naked plasmid VEGF 165 Intramuscular injection Phase I 2-11 months Improved Ankle-Brachial index, exercise time, new vessels on angiogram, limb salvage, improved tissue integrity... [Pg.319]

Subject A exercised his lower extremity muscles at home using a PC computer to control the implanted stimulator. In lanuary 1997, he was provided with a battery-operated external portable conditioning system (19x11x6 cm ), which he uses at home and at work sitting in his wheelchair. The exercise protocol stimulates the right and left knee extensors and ankle plantar/dorsi flexors alternately 4 sec ON/4 sec OFF, for a total of 20 min. After the muscles have been conditioned, dynamometric testing (isometric mode) has shown that implanted FES stimulation produces bilateral knee extension torque of 45 to 55 Nm at 30° and 65 Nm at 60° of knee flexion. Subject A exercised at least 3 days a week, and found if he did not do so the spasticity in the lower extremities increased. [Pg.530]

Hepatic myelopathy is characterized by a rapidly progressing spastic paraparesis without sensible deficits or bladder dysfunction. Brisk tendon reflexes and increased tone of the lower extremities are the predominant findings. Plantar reflexes may be flexor, even in patients with ankle clonus. Usually the patients are bound to a wheelchair or bedridden within months. [Pg.190]

Lower extremity prostheses are custom devices. While the connective componentry, joints (knees, feet/ankles), and suspension are commercially available, the socket that encases the residual limb is custom-designed. The socket design may or may not involve a computer, as in CAD-CAM. [Pg.898]

Prosthetic Feet. With the exception of partial foot amputees, the prostheses for all lower extremity amputees require a prosthetic foot. The prescription criteria for these feet take into consideration the amputation level, residual limb length, subject activity level, cosmetic needs, and the weight of the individual. Prosthetic feet range from the SACK (solid ankle cushioned heel) foot, which is relatively simple and inexpensive, to dynamic-response or energy-storing feet that are more complicated and considerably more costly. Note that prosthetic feet are often foot and ankle complexes. As such, prosthetic feet may replace plantarflexion/dorsiflexion, pronation/supination, and inversion/eversion. Prosthetic feet are typically categorized in terms of the function(s) they provide or replace and whether or not they are articulated. [Pg.900]

Although the preponderance of lower extremity involvement during acute episodes of gout was confirmed, the frequency of acute attacks at the first metatarsophalangeal was less than expected and knee and ankle involvement was more common than anticipated -Only 6U.20 % of patients had monoarticular gout in the first episode. The pattern of joint involvement was independent of duration of disease. [Pg.118]

Systolic pressures can be taken at different locations in the lower extremities to help identify the location of arterial disease. Most commonly, pressures are taken at the high thigh, lower thigh, calf, and ankle. [Pg.24]

Clearly, diabetics who have peripheral arterial disease have higher mortality rates than those who do not have arterial disease. A recent study by Vogt et al. (15) evaluated the relationship between peripheral arterial disease and mortality in a population of close to 2000 individuals over a 13-year period. All patients 50 years of age and older with no history of lower extremity surgery were evaluated for the presence of peripheral arterial disease. Analysis of the data stratified by populations and comorbid conditions showed that a low ankle-brachial index is an independent predictor of all causes of mortality in both men and women with peripheral arterial disease. This increase is a relative risk and is unchanged after exclusions of all patients with a clinical history of cardiovascular disease or diabetes. Thus, a low ankle-brachial index is an important measurement to obtain to assess the risk of mortality among those who smoke and have either angina or diabetes. [Pg.57]

Risk Factors of LEAD Co-Existing With Coronary Artery Disease Other Than Over 40 Years of Age What Do Patients With Lower Extremity Arterial Disease Assessed With a Low Ankle-Brachial Index Die From Dobutamine Stress Echocardiography Management... [Pg.183]

WHAT DO PATIENTS WITH LOWER EXTREMITY ARTERIAL DISEASE ASSESSED WITH A LOW ANKLE-BRACHIAL INDEX DIE FROM ... [Pg.188]

A 70-year-old woman presented with multiple symptoms of back and hip pain for 3 weeks. Although relevant somatic dysfunctions were noted to explain the chief symptom, examination of the lower extremities demonstrated + 2 pilling edema. The patient stated that she had had swollen ankles for several years and was on diuretics and calcium channel-blocking medication for this and her hypertension. The edematous legs made it difficult for her to walk comfortably the distances to the stores in her immediate neighborhood. She denied any history of myocardial infarction or shortness of breath. Other than the chief symptom, hypertension, and peripheral edema, there were no other medical problems. [Pg.597]

Injuries to the human lower extremities are mostly due to the collision of a vehicle and pedestrian, which results in fractures of long bones, injuries to the knee and ankle. This is because of the impact applied by the vehicle and high acceleration created in the lower extremities. Different injury mechanisms can be seen in long bones, which bending and torsional moments have been considered as major affecting factors [1]. [Pg.130]

Lower extremities, including hip, leg, ankle, foot and toes Other part of body Whole body and multiple sites... [Pg.59]


See other pages where Lower extremities ankle is mentioned: [Pg.140]    [Pg.226]    [Pg.160]    [Pg.396]    [Pg.400]    [Pg.186]    [Pg.86]    [Pg.530]    [Pg.901]    [Pg.672]    [Pg.884]    [Pg.906]    [Pg.909]    [Pg.22]    [Pg.150]    [Pg.10]    [Pg.13]    [Pg.24]    [Pg.25]    [Pg.25]    [Pg.299]    [Pg.597]    [Pg.573]    [Pg.981]    [Pg.162]    [Pg.565]    [Pg.966]    [Pg.220]    [Pg.133]    [Pg.282]    [Pg.212]    [Pg.57]   


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Ankle

Extreme

Extremities

Extremizer

Lower extremities

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