Inflammation cells

Houten SM, Frenkel J, Waterham HR (2003) Isoprenoid biosynthesis in hereditary periodic fever syndromes and inflammation. Cell Mol Life Sci 60 1118-1134... 

The activity of Bik affects various biological functions, including inflammation, cell signaling, and apoptosis. 

Quinlan TR, Berube KA, Marsh JP, et al. 1995. Patterns of inflammation, cell proliferation, and related gene expression in lung after inhalation of chrysotile asbestos. Am J Pathol 147 728-739. 

L. Osborn, Leukocyte Adhesion to Endothelium in inflammation. Cell, 62 (1990) 3-6. 

Teismann P, Schulz JB (2004) Cellular pathology of Parkinson s disease Astrocytes, microglia and inflammation. Cell Tissue Res 318 149-161. 

In order to develop meaningful assay systems to study mechanisms involved in chronic inflammation, cells from cartilage, bone, normal synovium, and from organized inflammatory lesions as well as leukocytes from spleen, thymus, and macrophages from serous cavities of laboratory animals maintained as stable populations in cell or organ culture have been utilized. 

Kaur T, Mukherjea D, Sheehan K, Jajoo S, Rybak LP, Ramkumar V (2011) Short interfering RNA against STAT1 attenuates cisplatin-induced ototoxicity in the rat by suppressing inflammation. Cell Death Dis 2 el80... 

Tissue inflammation Cell removal Morphological features Cellular membranes... 

The transcription factor nuclear factor kappa B (NF-kB) is a member of the Rel family of proteins that plays an important role in a variety of cellular response mechanisms including immunity, inflammation, cell growth, and apoptosis. Recent evidence suggests that this transcription factor is induced following exposure of cancer cells to apoptotic... 

The transcription factor NP-kB controls the expression of numerous genes that modulate the immune and stress responses, onset, and the resolution of inflammation, cell adhesion, calcium homeostasis, maintenance of intercellular communications, and regulation of cellular proliferation, and protection against... 

Cramer T, Yamanishi Y, Clausen BE, Forster I, Pawlinski R, Mackman N, Haase VH, Jaenisch R, Corr M, Nizet V, Firestein GS, Gerber HP, Ferrara N, Johnson RS. HIF-lalpha is essential for myeloid cell-mediated inflammation. Cell 2003 112 645-657. 

Molitemo AR, Resar LMS. AKNA another AT-hook transcription factor hooking-up with inflammation. Cell Res. 2011 21 1528-30. 

Gallic acid can reduce allergic symptoms by acting as an antihistamine. Recent investigations by Maggi-Capeyron et al. [100] have linked a series of phenolic acids with the inhibition of AP-1 transcriptional activity. AP-1 is an activator protein implicated in the processes that control inflammation, cell differentiation, and proliferation, so gallic acid is a potent anti-inflammatory agent. 

---