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Hyaline membrane

The proteolytic enzymes, trypsin, chymotrypsin, and chymoral [8076-22-0] in combination, have been used for the treatment of post-operative hand trauma, athletic injuries, and sciatica (214—216). Trypsin has also been used successfully in treating hyaline membrane disease of newborn babies, a condition usually fatal without treatment (217). Immobilized preparations of trypsin are useful in treating acute radiation cystitis following pelvic x-irradiation therapy (218). [Pg.312]

Abe, T. (1980). Artificial surfactant therapy in hyaline-membrane disease, Lancet. 1, 55-59. [Pg.321]

Fatty liver of pregnancy Placental abruption Preeclampsia/eclampsia Retained fetus syndrome Pulmonary syndrome syndrome Empyema Hyaline membrane disease... [Pg.996]

Infant respiratory distress syndrome (IRDS), also known as hyaline membrane disease, is one of the most common causes of respiratory disease in premature infants. In fact, it occurs in 30,000 to 50,000 newborns per year in the U.S. — most commonly in neonates bom before week 25 of gestation. IRDS is characterized by areas of atelectasis, hemorrhagic edema, and the formation of hyaline membranes within the alveoli. IRDS is caused by a deficiency of pulmonary surfactant. Alveolar type II cells, which produce surfactant, do not begin to mature until weeks 25 to 28 of... [Pg.248]

Barter RA, Byrne MJ, Carter RE. 1966. Pulmonary hyaline membrane Late results of injury to the lung linings. Arch Dis Child 41(219) 489-495. [Pg.166]

Stahlman, M. T. (1984). Chronic lung disease following hyaline membrane disease. In "Neonatal Medicine" (L. Stern and P. Vert, eds.), pp. 454-4 73. Masson, New York. [Pg.216]

Octreotide has been used in premature neonates for closure of enterocutaneous fistulae complicating necrotizing enterocolitis. Two cases of oxygen desaturation have been reported (12). The authors suggested that the effect may relate to pre-existing hyaline membrane disease. [Pg.503]

T. Fujiwara and F.H. Adams, Surfactant for hyaline membrane disease, Pediatrics 66 (1980) 795-798. [Pg.288]

The use of compressed oxygen can benefit not only athletes, but also vulnerable premature babies. Premature babies can be afflicted with hyaline-membrane disease. This condition prevents the alveoli in their lungs from inflating, which leads to serious breathing difficulties. Placing these babies in an oxygen-rich environment such as an HBO or an incubator (Figure 11.28) helps inflate the alveoli. This increases the infants chances for survival. [Pg.462]

Northway WH, Rosan RC, Porter DY. Pulmonary disease following respiration therapy of hyaline membrane disease. N Engl J Med 276 (1967) 357-368. [Pg.249]

Strange RC, Cotton W, Fryer AA, Jones P, Bell J, Hume R. Lipid peroxidation and expression of copper-zinc and manganese superoxide dismutase in lungs of premature infants with hyaline membrane disease and bronchopulmonary dysplasia. J Lab Clin Med 116 (1990) 666-673. [Pg.249]

Avery, M.E. Mead, J. Surface properties in relation to atelectasis and hyaline membrane disease. Am. J. Dis. Child. 1959, 97, 517-526. [Pg.3596]

Systemic Rat 1 or 2 hr (Wistar) Resp 27 M (death by asphyxiation lung edema hyaline membranes, necrosis of alveolar epithelium and fibrosis) Livardjani et al. 1991 ELEM... [Pg.55]

Respiratory effects in animals have been observed following acute inhalation exposure of metallic mercury vapors. Rats exposed to 27 mg/m3 of elemental mercury vapors for 2 hours then observed for 15 days displayed dyspnea and death due to asphyxiation (Livardjani et al. 1991b). Respiratory tract lesions included lung edema, necrosis of the alveolar epithelium and hyaline membranes, and occasional lung fibrosis. [Pg.64]

The use of a biexponential equation with postnatal age as the time scale permits some practical interpretation of the time course component of the final PD model. Table 27.2 presents the peak spell frequency, the time to achieve peak frequency, and the model predicted resolution half-time of apnea in absence of therapy. The resolution half-time defines the number of days of postnatal maturation that transpire before the daily spell frequency is reduced by one-half. The influence of hyaline membrane disease on resolution half-time is readily apparent. The most premature neonates with HMD have the slowest time to maximum episode counts and have the highest frequency of apnea. A 24 week gestational age infant with HMD requires an additional 7 days for a maturational reduction in spell count of one-half. The half-time of apnea onset is approximately 2.5 days. On average, the greatest severity of apnea would occur at approximately 1 postnatal week. Figure 27.10 depicts the baseline apneic episode frequency versus postnatal age for each gestational age in the present study. The predictions of daily spell count are population predictions, calculated using the final parameter estimates for PRE, and... [Pg.715]

The predicted time to maximum severity (t ax), the expected maximum number of spells per day, and the resolution half-time were calculated from the final PD model parameter estimates. These figures represent the anticipated disease severity and time course in the absence of theophylline therapy. HMD = hyaline membrane disease. [Pg.716]

Postexposure survival remains a perplexing problem. In the early work, death was probably due to many factors such as exhaustion from the work of breathing, contamination of lungs by urine, feces, and skin oils, atelectasis due to loss of pulmonary surfactant, hyaline membrane, and possibly electrolyte imbalances, especially in the lung parenchyma. However, Kylstra and Lanphier devised techniques to obviate the difficulties en-... [Pg.93]

B23. Berfenstam, B., Edlund, T., and Zettergren, L., The hyaline membrane disease, a review of earlier clinical and experimental findings and some studies on the pathogenesis of hyaline membranes in Oa-intoxicated rabbits. Acta Paediat. 47, 82-100 (1958). [Pg.123]

RDS, historically known as hyaline membrane disease (HMD), is more appropriately termed surfactant-deficiency RDS. RDS is associated with considerable morbidity and mortality. Before 35 weeks gestation, the risk of RDS and the severity of disease increase with greater degree of prematurity and, in the absence of appropriate antenatal interventions, occurs in over 50% of newborns of 30 weeks or less gestation. The Vermont Oxford Network experience for 1999 describes over 27,000 neonates below 1500 g from 325 neonatal intensive care unit (NICU) sites. The annual report noted that RDS occurred in over 80% of premature infants below 1000 g and that there was a gradual decline to about 42% of neonates with birth weights between 1400 and 1500 g. [Pg.557]


See other pages where Hyaline membrane is mentioned: [Pg.187]    [Pg.47]    [Pg.250]    [Pg.291]    [Pg.250]    [Pg.586]    [Pg.28]    [Pg.39]    [Pg.159]    [Pg.166]    [Pg.16]    [Pg.60]    [Pg.209]    [Pg.498]    [Pg.242]    [Pg.449]    [Pg.451]    [Pg.1452]    [Pg.2166]    [Pg.2197]    [Pg.710]    [Pg.711]    [Pg.75]    [Pg.79]    [Pg.93]    [Pg.96]   
See also in sourсe #XX -- [ Pg.100 ]

See also in sourсe #XX -- [ Pg.191 ]




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