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Gene expression reprogramming

Izumo, S., Nadal, G.B., Mahdavi, V. (1988). Protooncogene induction and reprogramming of cardiac gene expression produced by pressure overload. Proc. Natl. Acad. Sci. USA 85, 339-343. [Pg.455]

Mechanism of Action Lithium s pharmacologic mechanism of action is not well understood and probably involves multiple effects. Possibilities include altered ion transport, increased intraneuronal catecholamine metabolism, neuroprotection or increased brain-derived neurotrophic factor, inhibition of second messenger systems, and reprogramming of gene expression.29... [Pg.592]

Lindbo, J.A., Fitzmanrice, W.P., and della-Cioppa, G. (2001). Virus-mediated reprogramming of gene expression in plants. Curr. Opin. Plant Biol. 4, 181-185. [Pg.94]

THE REGULATION AND REPROGRAMMING OF GENE EXPRESSION IN THE PREIMPLANTATION EMBRYO... [Pg.129]

V. Role of DNA Replication in Reprogramming Gene Expression during ZGA. . . 140... [Pg.129]

V. ROLE OF DNA REPLICATION IN REPROGRAMMING GENE EXPRESSION DURING ZGA... [Pg.140]

Davis, W., Jr., and Schultz, R.M. (1997). Role of the first round of DNA replication in reprogramming gene expression in the preimplantation mouse embryo. Mol. Reprod. Dev. 47 430-434. [Pg.160]

Since there is an absence of reports detailing the activation of human SCNT oocytes and the successful production of cloned blastocysts, it was necessary to identify several parameters, including the reprogramming time (the time between cell fusion and egg activation, returning the gene expression profile of the somatic cell to that needed for appropriate embryonic development) and activation methods. Based on results from animal SCNT oocytes and the parthenogenetic activation of human oocytes, we initially employed a porcine activation protocol (simultaneous fusion and activation with electrical pulse) which used hu-... [Pg.282]

The clinical efficacy of lithium in the prophylaxis of recurrent affective episodes in bipolar disorder is characterized by a lag in onset and remains for weeks to months after discontinuation. Thus, the long-term therapeutic effect of lithium likely requires reprogramming of gene expression. Protein kinase C and GSK-3 signal transduction pathways are perturbed by chronic lithium at therapeutically relevant concentrations and have been implicated in modulating synaptic function in nerve terminals (84). [Pg.873]

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See also in sourсe #XX -- [ Pg.132 , Pg.140 , Pg.141 , Pg.142 , Pg.143 ]



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Reprogramming

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