Fatty acids fecal

The fecal flora ferment unabsorbed carbohydrates with production of short-chain fatty acids (SCFAs). One likely explanation for diarrhea due to antibiotics is altered fecal... 

The daily output of fecal fat may be regarded as the simplest quantitative measure of the effect of gluten on these patients. The evidence available suggests that the extra fat is derived from dietary fat and mainly represents interference with absorption. There is usually a disproportionate increase of saturated as compared with unsaturated fats (W4). The reason for this is not clear. There may be selective rejection of longer chain and more saturated fats there may also be increased hydrogenation of unsaturated fatty acids (SI). The increase of fat output may occur within a day or 2, or it may be delayed for 10 days or more. There are many possible explanations for this delay. It may require many small insults to the mucous membrane... 

As shown in Table III, mean fecal calcium losses tended to be higher when the higher fat diet was fed in comparison to results when the lower fat diet was fed. Therefore, apparent calcium absorption was higher when the low fat diet was fed. These differences were significant at only the P< 0.075 level hence, only a trend was illustrated. In this study no attempt was made to equalize fatty acid proportionality patterns or cholesterol intake. These or other dietary or non-dietary factors may have influenced the observed apparent trends. Other studies with human adults have not demonstrated any apparent influence on level of dietary fat on calcium absorption. 

Bourquin LD, Titgemeyer EC, Fahey GC Jr. Vegetable fiber fermentation by human fecal bacteria cell wall polysaccharide disappearance and short-chain fatty acid production during in vitro fermentation and water-holding capacity of unfermented residues. J Nutr 1993 123 860-869.. 

Fecal steroid and lipid excretion. Fiber supplements from sugar cane residue (bagasse), administered to volunteers for 12 weeks, increased stool weights and stool fat excretion. Bagasse increased the daily loss of acid steroids and decreased transit time without alteration in fecal flora. The increased excretion of bile acids and fatty acids failed to lower the plasma cholesterol and TGs after 12 weeks ". 

Pigs dosed with radiolabeled avilamycin produced three unidentified fecal metabolites derived from the oligosaccharide and/or the eurekanate moieties. However, the primary metabolite in feces and liver was flambic acid. Mean total residues in muscle were all below 0.2 ppm, whereas residues in other edible tissues were all below 1 ppm. Most of tissue residues were derived from the oligosaccharide and/or the eurekanate portion of avilamycin, whereas very little was parent avilamycin. In fat, the avilamycin related residues was found to be due to radioactivity that had entered normal metabolic pathways and had been incorporated into the fatty acids. 

There are several ways pectin could reduce serum cholesterol. In studies with human subjects, fecal excretion of bile acids, fatty acids, and total steroids increased when subjects were fed 15-40 g/day of pectin (58, 63, 64). Since pectin usually lowers serum cholesterol only when cholesterol is present in the diet, it seems that pectin might act by reducing cholesterol absorption. Several groups have found that in rats dietary... 

Kotchan, A.B. and Baidoo, S.K., The effect of Jerusalem artichoke supplementation on growth performance and fecal volatile fatty acids of grower-finisher pigs, Can. Soc. Sci. Ann. Conf. Proc., 223, 97T-25, 1997. 

Hamm CE, Reigstad M, Wexels Riser C, Miihlebac A, Wass-mann P (2001) On the trophic fate of Phaeocystis pouchetii. VII. Sterols and fatty acids reveal sedimentation of P. puchelii-dcnved organic matter via krill fecal strings. Mar Ecol Prog Ser 209 55-69... 

In Study II, young adult human subjects were fed two laboratory-controlled diets containing either 30% of total calories from fat (approximately 100 mg cholesterol 10 10 10 PUFA to MUFA to SFA ratio) or 40% of total calories from fat (approximately 600 mg cholesterol 4 14 14 fatty acid ratio). Two levels of manganese were fed (5 and 45 mg Mn/day) within each level of fat. The higher level of dietary fat generally increased fecal excretion of manganese and increased serum lipids. 

Enrichment of the dietary fat with PLs or TAGs may or may not affect fecal excretion of fat and minerals and may increase or decrease saturated fat absorption depending on the PL and TAG source (83). Long-chain polyunsaturated fatty acids were better absorbed in preterm infants when fed as PLs than as TAGs (84). Feeding long-chain polyunsaturates as PLs or TAGs influences the distribution of these fatty acids in plasma lipoprotein fractions, affects their content in different plasma... 

Fatty acid synthetase, 183 Fatty acid transport, 215-216,12D, 777-774 Falty liver, 293 alcoholism, 250-251 choline deficiency, 3l7 Fatty streak, 360, 636 Fecal blood test, 84 Feedback inhibition, 256 Feeding center, brain, 103-104 Fenton reaction, 627.635,903 Fermentative metabolism, 159,181-182, 233-243... 

Short-chain fatty acids, especially butyrate, are a preferred source of energy for the colonic epithelium. There is evidence to suggest that butyrate enemas are effective in the treatment of ulcerative cohtis. The seeds of Plantago ovata (a source of fermentable dietary fiber) increase fecal concentrations of butyrate and acetate. In a randomized, open, parallel-group, multicenter study in 105 patients with ulcerative cohtis, P. ovata seeds 10 mg bd were as effective as mesalazine 500 mg tds in maintaining remission over 12 months (10). Adverse effects were similar in the two groups, and included constipation, flatulence, nausea, and diarrhea. 

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