Exuded cytoplasm

The second set of experiments used exuded cytoplasm from Chara. Here, streaming was totally inhibited by the addition of CB, and although the fibrils were still present, their motility was significantly reduced relative to controls. Notably, Characean microfilaments are uncharacteristic in that they are operationally different.41 They fail to shorten to change cell morphology but give rise to a motile force that is parallel to their surface, thereby creating movement.43... 

Chlamydia 5-10 days Diffuse hyperemia, purulent exudate Basophilic cytoplasmic inclusion bodies... 

In the hamster, multiple short-term intratracheal instillations of zirconium lactate produced lesions beginning with exudative pneumonia followed by pneumonitis (interstitial pneumonia) and foreign body granulomas. Electron microscope microprobe analysis demonstrated the metallic component of the instilled compound in membrane-bound cytoplasmic inclusions of macrophages (Leininger et al. 1977). 

During the earliest stages, the first cells to react against the tubercle bacilli are the polymorphonuclear leucocytes, which encompass and phagocytose the organisms (Vorwald 1931). After intravenous injection into rabbits, this reaction was confined at first to small capillaries, and then extended to adjoining alveolar walls and spaces. Within 14 h, the polymorphonuclear leucocytes with and without tubercle bacilli were phagocytosed by the mononuclear exudate cells, which increased in number to the exclusion of the leucocytes. The tubercle bacilli thus were liberated in the cytoplasm of the exudate cell. 

During the 1950s, an interrelationship between selenium and vitamin E was established. It was found that selenium prevented exudative diathesis in vitamin E-deficient chicks and liver necrosis in vitamin E-deficient rats. Subsequent research demonstrated that both selenium and vitamin E protect the cell from the detrimental effects of peroxidation, but each takes a distinctly different approach to the problem. Selenium functions throughout the cytoplasm to destroy peroxides, while vitamin E is present in the membrane components of the cell and prevents peroxide formation. This explains why the biological need for each nutrient can be offset, at least partially although not totally, by the other. 

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