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Visual evoked potential

Other antiepileptic drugs can also alter visual evoked potentials and brainstem evoked potentials. Visual field defects associated with various antiepileptic drugs (carba-mazepine, diazepam, gabapentin, phenytoin, tiagabine, and vigabatrin) have been reviewed (37). [Pg.3628]

An increased level of exploratory activity immediately after exposure, attributed to reduced anxiety on the part of the rats, was also observed in this study. Decreased avoidance was observed in rats exposed to 125 ppm trichloroethylene 4 hours per day, 5 days per week for 30 days (Goldberg et al. 1964a). Changes in visually evoked potentials (Blain et al. 1992) and electroretinal responses to flash stimulation (Blain et al. 1994) were seen in rabbits exposed to 350 ppm trichloroethylene for 12 weeks (4 days/week, 4 hours/day). The study authors suggested that binding of trichloroethanol to blood proteins may enable it to reach the visual cortex. [Pg.54]

Dissemination in space by MRI evidence of nine or more T2-weighted brain lesions, or two or more cord lesions, or four to eight brain and one cord lesion, or positive visual evoked potentials with four to eight MRI lesions, or positive visual evoked potentials with less than four brain lesions plus one cord lesion. [Pg.434]

NS (children, environmental exposure) Neurological Blood lead correlated with alterations in visual evoked potentials range, 1.4-17.4 Altmann et al. 1998 Winneke et al. 1994... [Pg.43]

Ewert T, Beginn U, Winneke G, et al. 1986. [Sensory nerve conduction and visual and somatosensory evoked potential in children exposed to lead.] Nervenarzt 57 465-471. (German)... [Pg.519]

Winneke G. 1980. Non-recovery of lead-induced changes of visual evoked potentials in rats. Toxicol Lett 1 77. [Pg.586]

Di Piazza et al. 51 Neomycin (double-blind) 7 days Flapping tremor, bradylalia, patient self-evaluation, visual evoked potential reaction, trial making test E Rif > Neo T Rif = Neo... [Pg.93]

Zeneroli ML, Ventura E, Baraldi M, Penne A, Messori E, Zieve L Visual evoked potentials in encephalopathy induced by galactosamine, ammonia, dimethyldisulfide and octanoic acid. Hepatology 1982 2 532-538. [Pg.94]

Araki, S, Murata, K, Aono, H. (1987). Central and peripheral nervous system dysfunction in workers exposed to lead, zinc and cooper. A follow-up study of visual and somatosensory evoked potentials. Int. Arch. Occup. Environ. Health, 59, 177-87. [Pg.423]

Seppalainen AM, Raitta C, Huuskonen MS. 1979. -Hexane-induced changes in visual evoked potentials and electroretinograms of industrial workers. Electroencephal Clin Neurophysiol 47 492-498. [Pg.246]

P. A. Bhaskar, S. Vanchilingam, E. A. Bhaskar, A. Devaprabhu, and R. A. Ganesan. Effect of L-dopa on visual evoked potential in patients with Parkinson s disease. Neurology 36 1119-1121 (1986). [Pg.338]

Reeves RR, Struve FA, Patrick G. (1999). The effects of caffeine withdrawal on cognitive P300 auditory and visual evoked potentials. Clin Electroencephalogr. 30(1) 24-27. [Pg.461]

The Neurophysiology Sensory Evoked Potentials test guideline (OPPTS 870.6855) is designed to detect and characterize changes in the sensory aspects of nervous system function that result from exposure to chemical substances. The techniques involve neurophysiological measurements from adult animals and are sensitive to changes in the function of auditory, somatosensory (body sensation), and visual sensory systems. [Pg.132]

Lund SP, Simonsen L, Fries AS Two weeks inhalation exposure to 4- tt-butyltoluene causes persistent changes in visually evoked potentials in rats. Pharmacol Toxicol 76(1) 36-40, 1995... [Pg.108]

Figure 3.5 Classification of the components of visually evoked potentials (VEPs). The two negative peaks I and III and the positive peak II were termed the primary response by Ciganek (1961), peaks IV-VII (negative peaks = upwards pointing peaks) being termed the secondary response. The amplitude of these peaks lies in the region of 10-30/iV. In addition to amplitudes (e.g. between peaks I and II), latencies are determined, i.e. the time in milliseconds between the stimulus and the appearance of a peak, e.g. peak IV... Figure 3.5 Classification of the components of visually evoked potentials (VEPs). The two negative peaks I and III and the positive peak II were termed the primary response by Ciganek (1961), peaks IV-VII (negative peaks = upwards pointing peaks) being termed the secondary response. The amplitude of these peaks lies in the region of 10-30/iV. In addition to amplitudes (e.g. between peaks I and II), latencies are determined, i.e. the time in milliseconds between the stimulus and the appearance of a peak, e.g. peak IV...
Lanting, P. et al., The cause of increased pupillary light reflex latencies in diabetic patients the relationship between pupillary light reflex and visual evoked potential latencies, EEG Clin. Neurophysiol., 78, 111, 1991. [Pg.142]

Cortical sources of the early components of visual evoked potential. Hum Brain Mapp 15 95-111. [Pg.225]

List of Abbreviations ISI, interstimulus interval LGN, lateral geniculate nucleus M, magnocellular MST, medial superior temporal area MT, medial temporal area NMDA, N-methyl-D-aspartate P, parvocellular ssVEP, steady-state visual evoked potential TE, temporal lobe TEO, temporal occipital area tVEP, transient visual evoked potential YEP, visual evoked potential VI, primary visual cortex... [Pg.332]


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See also in sourсe #XX -- [ Pg.75 ]




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Electrophysiological measures visual evoked potentials

Evoked

Evoked potentials

Pattern-reversal visual evoked potentials,

Sweep visual evoked potential

Visual evoked potential and

Visual evoked potentials animal studies

Visually evoked potential

Visually evoked potential

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