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ER, Estrogen receptor

Fig. 4.1. Cellular model illustrating cell types in vascular wall involved in vasorelaxation induced by SERMs. Putative targets of SERMs are indicated within cyan tags. SERMs directly affect L-type VDCC, BK fil subunit in smooth muscle cells, and ER in endothelial cells. L-type VDCC L-type voltage-dependent calcium channel BK calcium-activated large conductance K+ channel PKG protein kinase G eNOS endothelial nitric oxide synthase GC soluble guanylate cyclase cGMP cyclic GM P V electrochemical membrane potential ER estrogen receptor. See text for further details... Fig. 4.1. Cellular model illustrating cell types in vascular wall involved in vasorelaxation induced by SERMs. Putative targets of SERMs are indicated within cyan tags. SERMs directly affect L-type VDCC, BK fil subunit in smooth muscle cells, and ER in endothelial cells. L-type VDCC L-type voltage-dependent calcium channel BK calcium-activated large conductance K+ channel PKG protein kinase G eNOS endothelial nitric oxide synthase GC soluble guanylate cyclase cGMP cyclic GM P V electrochemical membrane potential ER estrogen receptor. See text for further details...
ER Estrogen Receptor, AR Androgen Receptor, GR Glucocorticoid Receptor, TR Thyroid Receptor, RAR Retinoic Acid Receptor, RXR Retinoie X Receptor, PR Progesterone Receptor, VDR Vitamin D Receptor, PPAR Peroxisome Proliferator-Activated Receptor, PXR Pregnane X Receptor, ROR Retinoic Acid-Related Orphan Receptor, NHF-4 Hepatocyte Nuclear Eactor 4, ERR Estrogen-Related Receptor... [Pg.2]

Jensen EV, DeSombre ER. Estrogen-receptor interaction. Science 1973 182 126-134. [Pg.713]

Figure 23-8 Functional domains of the steroid hormonere ceptors. AR, androgen receptor ER. estrogen receptor. GR, glucocorticoid receptor. MR. mineralocorlicoid receptor and F progesterone receptor. Figure 23-8 Functional domains of the steroid hormonere ceptors. AR, androgen receptor ER. estrogen receptor. GR, glucocorticoid receptor. MR. mineralocorlicoid receptor and F progesterone receptor.
RBA, relative binding affinity % of estradiol activity) ER, estrogen receptor. [Pg.1066]

CMF cyclophosphamide, methotrexate, fluorouracil (regimen) DCIS ductal carcinoma in situ DPS disease-free survival ER estrogen receptor... [Pg.2361]

ACTH, adrenocorticotropin a-su, alpha subunit -end, beta-endorphin p-FSH, beta follicle stimulating hormone fS-LH, beta luteinizing hormone -LPH, beta lipotropin -TSH, beta thyroid stimulating hormone, ER, estrogen receptor GH, growth hormone. [Pg.296]

TTF-1, thyroid transcription factor-1 CK, cytokeratin ER, estrogen receptor, PR, progesterone receptor PSA, prostate-specific antigen PAP, prostatic acid phosphatase. [Pg.325]

CR, calretinin CK5/6, cytokeratin 5/6 ER, estrogen receptors. +, almost always positive N, negative. [Pg.743]

Figure 16.7 Immunohistochemical staining intensity as a function of the duration of formahn fixation. Immunostaining intensity was measured in triphcate.The coefficients of variation (CVs) were almost aU less than 10% and, for purposes of clarity, are not shown. Staining intensity is measured as mean pixel intensity units on a 1-256 scale. ER(1D5), ER(2-123), PR(636), PR(1294), and Ki-67(MIB-1), DakoCytomation, Carpinteria, CA PR(1A6) and HER2(CB11), Vision Biosystems, NorweU, MA. ER, estrogen receptor PR, progesterone receptor. Reproduced with permission from Reference 15, 2006 American Society for Clinical Pathology. Figure 16.7 Immunohistochemical staining intensity as a function of the duration of formahn fixation. Immunostaining intensity was measured in triphcate.The coefficients of variation (CVs) were almost aU less than 10% and, for purposes of clarity, are not shown. Staining intensity is measured as mean pixel intensity units on a 1-256 scale. ER(1D5), ER(2-123), PR(636), PR(1294), and Ki-67(MIB-1), DakoCytomation, Carpinteria, CA PR(1A6) and HER2(CB11), Vision Biosystems, NorweU, MA. ER, estrogen receptor PR, progesterone receptor. Reproduced with permission from Reference 15, 2006 American Society for Clinical Pathology.
AR Androgen receptor ER Estrogen receptor FLIP Fluorescence loss in photobleaching FRAP Fluorescence recovery after photobleaching FRET Fluorescence resonance energy transfer GR Glucocorticoid receptor NER Nucleotide excision repair UV Ultra-violet... [Pg.178]

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See also in sourсe #XX -- [ Pg.559 , Pg.902 ]



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