Cyanide seizures caused

Estimates of toxicity based on accidental and deliberate poisoning in humans indicates that NaCN has a lethal potential of around 2-3 mg kg 1 and KCN at 3-5 mg kg 1 (Ballantyne, 1987b). Ingestion of alkaline CN salts by humans (calcium, sodium and potassium cyanides) may cause symptomatology which is life-threatening in 30 to 60 min (Hall and Rumack, 1998). Rapid progression to coma, seizures, arrhythmias, intractable hypotension and apnoea may occur, and death is common. 

Cyanide exposure can cause transient CNS stimulation, followed by syncope, ataxia, dyspnea, seizures, paralysis. 

The seizure of cyanide ammunitions, in spite of the economical consequences for the FARCs could be seen connected with the will of cause as psychological effects on the governmental troops. 

Finally, cyanides such as hydrogen cyanide (AC) and cyanogen chloride (CK) both release cyanide ions in the body. Lower doses cause headaches, weakness, disorientation, and nausea higher doses cause circulatory effects, seizures, and respiratory and cardiac failure. While often attributed to its blockade of energy metabolism, the mechanisms of cyanide intoxication remain unclear and may include cellular targets more sensitive to inhibition than cytochrome oxidase. 

B. Asphyxia and systemic intoxicants may cause dizziness, confusion, syncope, seizures and coma. In addition, carbon monoxide (see p 151), cyanide poisoning (p 177), and methemoglobinemia (p 261) have been documented in victims of smoke inhalation. 

D. Cyanides cause dizziness, dyspnea, confusion, agitation, and weakness, with progressive obtundation and even coma. Seizures and hypotension followed by cardiovascular collapse may occur. 

Cyanide causes gasping for air red eyes, skin, and lips seizures unconsciousness and death. 

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