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Copper recess

RoUs and other relatively simple shapes make use of inert shields and thieves to avoid edge buildup and produce a more even plate thickness. For more compUcated shapes having deeper recesses thicker deposits from cyanide copper baths have been used as an undercoat to the copper sulfate deposit. Acid copper baths operate near 100% efficient over a wide current density range. The cathode efficiency is usuaUy slightly less than the anode efficiency, bringing about a slow increase in copper unless drag-out losses are high. [Pg.158]

A more dilute strike bath is employed for obtaining the initial deposit on steel, while for strongly recessed parts, e.g. tubular work, an immersion nickel deposit has been used. A short cyanide copper strike is used before plating on zinc-base die castings. [Pg.519]

The copper cyanide bath has excellent macro-throwing power and is chosen whenever irregular-shaped parts are to be plated. The sulphate bath is not inferior when parts with very narrow recesses, i.e. with width of opening less than 6 mm, are to be plated, although its macro-throwing power is... [Pg.519]

Wilson s disease is another autosomal recessive disease leading to cirrhosis. Protein abnormalities result in excessive copper deposition in body tissues. The faulty protein is responsible for facilitating copper excretion in the bile, so copper accumulates in hepatic tissue. High copper levels within hepatocytes are toxic, and fibrosis and cirrhosis may develop in untreated patients. Those with Wilson s disease usually present with symptoms of liver or neurologic disease while still in their teens. [Pg.329]

Wilson s disease is an autosomal recessive disorder characterized by the accumulation of copper in liver and brain [21]. Hepatic involvement may result in liver cirrhosis and hepatic cancer. The deposition of copper in the basal ganglia results in a variety of movement disorders, including... [Pg.773]

Two inherited human diseases that represent abnormal copper metabolism are Menkes syndrome and Wilson s disease. Menkes syndrome, with symptoms similar to those of copper deficiency, is characterized by a progressive brain disease, abnormally low copper concentrations in liver and other tissues, and diminished ability to transfer copper across the absorptive cells of the intestinal mucosa (USEPA 1980 Aaseth and Norseth 1986). Wilson s disease (hepatolenticular degeneration) is the only significant example of copper toxicity in humans. Wilson s disease is an autosomal recessive disorder that affects normal copper homeostasis and is characterized by excessive... [Pg.134]

Zinc is used for a variety of indications. Zinc acetate (8.102) or, rarely, zinc sulfate (8.103) have been used orally to treat Wilson s disease, a recessively inherited disorder of copper metabolism, characterized by brain and liver dysfunction arising from excessive deposits of copper. Zinc pyrithione (8.104) is used in shampoos to treat seborrhea. Zinc propionate (8.105) and zinc caprylate (8.106) have been used as topical antifungal agents. [Pg.535]

Type 98 No 7 Mk 6 Model 1 Bomb contd four Thermite-filled electron fire pots. A central channel filled with quick match ran thru the length of the bomb. A BkPdr chge was located in the circular recess of the nose piece (pp 71—72) Type 98 No 7 Mk Model 2 Bomb contd a central Thermite core surrounded by a solidified kerosene, petrol, alcohol, soap mixture. A BkPdr burster chge was located in the nose. A copper tube contg quickmatch was located inside Thermite tube (pp 73—4) ... [Pg.465]

Copper Essential to all organisms constituent of redox enzymes and hemocyanin." Very toxic to most plants highly toxic to Invertebrates, moderately so to mammals. Pollution from industrial smoke and possibly from agricultural use. Wilson s disease, genetic recessive, results in toxic increase in copper storage. [Pg.485]

Wilson s disease, an autosomally recessively inherited disorder, is due to a defect in copper metabolism which leads to an accumulation of the element in the liver. A subse-... [Pg.94]

The way of connecting the current conductors to electrodes which remain permanently dipped into the solution of electrolytes depends on the material of the electrode (whether noninetallic, such as graphite, or metallic.) One possible method of connecting a graphite rod to the current conductor is schematically represented in Fig. 41. the upper end of the electrode has a small recess it is encompassed by a two-piece copper terminal which is connected to the current conductor. A terminal for connection of a plate shaped electrode to the current conductor is illustrated in Fig. 42. At first the upper edge of plate A is fitted... [Pg.183]

Wilson s disease Autosommal recessive, low serum ceruloplasmin and increased hepatic copper content. [Pg.136]

Cause/Inddence Genetic disorder, first described by Menkes in the early 1960s, resulting in problems with copper distribution through the body. The genetic defect is X-linked recessive, meaning that males (XY) inherit the defect and are affected, while females (XX) may be affected but are more likely to be carriers. While this condition can be inherited, as much as one-third of cases may arise as a result of a new gene mutation. Estimated incidence is somewhere between 1 in 30000 and 1 in 250000 individuals. [Pg.67]

Cause/Incidence Hereditary autosomal recessive genetic disorder first described by Wilson in 1912, which gives rise to altered copper transport resulting in excess metal storage. This condition is more commonly observed in males, with an incidence of around 1 in 30 000, and it is believed 1 in 100 individuals are imaffected carriers. [Pg.68]

Caeruloplasmin Copper-incorporating a glycoprotein true function remains unclear but acts as a copper donor and oxidative enzyme Low caeruloplasmin levels may be seen in cirrhosis (especially primary biliary cirrhosis) as caeruloplasmin is excreted hepatically Levels increased in infection, injury or inflammation. Low levels are found in Wilson s disease, which is an autosomal recessive disorder of copper metabolism it results in copper deposition in the liver, basal ganglia and eyes, and culminates in cirrhosis and neurological impairment... [Pg.26]


See other pages where Copper recess is mentioned: [Pg.539]    [Pg.539]    [Pg.126]    [Pg.385]    [Pg.145]    [Pg.452]    [Pg.264]    [Pg.587]    [Pg.17]    [Pg.733]    [Pg.774]    [Pg.172]    [Pg.321]    [Pg.155]    [Pg.126]    [Pg.172]    [Pg.618]    [Pg.1004]    [Pg.327]    [Pg.145]    [Pg.456]    [Pg.836]    [Pg.653]    [Pg.438]    [Pg.70]    [Pg.71]    [Pg.187]    [Pg.418]    [Pg.419]    [Pg.449]    [Pg.132]    [Pg.321]    [Pg.956]   
See also in sourсe #XX -- [ Pg.539 ]




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