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Cellular Calcium

Shimomura, O., Musicki, B., Kishi, Y., and Inouye, S. (1993a). Light-emitting properties of recombinant semi-synthetic aequorins and recombinant fluorescein-conjugated aequorin for measuring cellular calcium. Cell Calcium 14 373-378. [Pg.438]

Na/K Pump Activity and its Role in Cellular Calcium Homeostasis... [Pg.53]

Coetzee and Opie (1988, 1992) have suggested that the cellular calcium overload induced by oxidant stress is mediated by an increase in calcium influx through the L-type calcium channel. However, a number of other studies, using different radical-generating systems, experimental conditions and species, have described no significant effects on the calcium channel over the period of exposure necessary to induce cellular calcium overload (Bhatnagar etal., 1990 Shattock etal., 1990 Beresewicz... [Pg.60]

The sarcolemmal Na/K pump plays an imp>ortant, although indirect role in the regulation of cellular calcium homeostasis. The transmembrane Na gradient is maintained by the activity of the Na/K pump and the thermodynamic energy of this gradient in turn drives the Na/Ca exchange mechanism (Sheu and Fozzard, 1982 Barry and Bridge, 1993). Thus, the intracellular Ca concentration is closely related to intracellular Na and the activity of the Na/K pump (Bers and Ellis, 1982). [Pg.61]

Figure 4 Schematic representation of the Ca2+-transporting systems affecting cellular calcium homeostasis during hormonal stimulation, oq = oq-adrenergic receptor VP = vasopressin receptor PLC = phospholipase C PI = phosphatidylinositol PIP = phospha-tidylinositol-4-phosphate PIP2 = phosphatidylinositol-4,5-biphosphate IP3 = inositol-1,4,5-triphosphate DG = diacylglycerol PKC = protein kinase C. (Modified from Refs. 125 and 285.)... Figure 4 Schematic representation of the Ca2+-transporting systems affecting cellular calcium homeostasis during hormonal stimulation, oq = oq-adrenergic receptor VP = vasopressin receptor PLC = phospholipase C PI = phosphatidylinositol PIP = phospha-tidylinositol-4-phosphate PIP2 = phosphatidylinositol-4,5-biphosphate IP3 = inositol-1,4,5-triphosphate DG = diacylglycerol PKC = protein kinase C. (Modified from Refs. 125 and 285.)...
Takeshima H, Komazaki S, Nishi M, lino M, Kangawa K 2000 Junctophilins A novel family of junctional membrane complex proteins. Mol Cell 6 11-22 van Breemen C, Farinas BR, Gerba P, McNaughton ED 1972 Excitation-contraction coupling in rabbit aorta studied by the lanthanum method for measuring cellular calcium influx. Circ Res 30 44-54... [Pg.41]

Carafoli E, Santella L, Branca D, Brini M 2001 Generation, control, and processing of cellular calcium signals. Crit Rev Biochem Mol Biol 36 107—260 Chen Q, van Breemen C 1993 The superficial buffer barrier in venous smooth muscle sarcoplasmic reticulum refilling and unloading. Br J Pharmacol 109 336—343 Clapham DE, Runnels LW, Strubing C 2001 The TRP ion channel family. Nat Rev Neurosci 2 387-396... [Pg.136]

Griffin, D.S., and SegaU, H.J., 1987, Role of cellular calcium homeostasis in toxic liver injury induced by the pyrrolizidine alkaloid senecionine and the alkenal trans-4-OH-2-hexenal,... [Pg.144]

Gussone N, et al. (2006) Cellular calcium pathways and isotope fractionation in Emiliania huxleyi. Geology 34 625-628... [Pg.246]

Selected entries from Methods in Enzymology [vol, page(s)] Cellular calcium determination, 238, 73, 146, 298 calibration,... [Pg.303]

Fig. 2. Effect of calcium antagonists (CA) on a cardiac cell. Top typical cardiac action potential. The calcium (slow) inward current flows during the characteristic plateau phase (phase 2) of the action potential. This calcium influx is selectively inhibited by CA. Activation of the sarcoplasmic reticulum (SR) and other cellular calcium pools occurs via Ca + and Na+ ions which flow into the cell. The SR and other pools donate activator Ca + ions which stimulate the contractile proteins. The presence of tubular systems (invaginations), which are characteristic of cardiac tissues, results in considerable enlargement of the cellular surface, thus enabling an effective influx of Na+ and Ca + ions. Inhibition of the calcium inward flux by a CA causes diminished activation of the contractile proteins. Fig. 2. Effect of calcium antagonists (CA) on a cardiac cell. Top typical cardiac action potential. The calcium (slow) inward current flows during the characteristic plateau phase (phase 2) of the action potential. This calcium influx is selectively inhibited by CA. Activation of the sarcoplasmic reticulum (SR) and other cellular calcium pools occurs via Ca + and Na+ ions which flow into the cell. The SR and other pools donate activator Ca + ions which stimulate the contractile proteins. The presence of tubular systems (invaginations), which are characteristic of cardiac tissues, results in considerable enlargement of the cellular surface, thus enabling an effective influx of Na+ and Ca + ions. Inhibition of the calcium inward flux by a CA causes diminished activation of the contractile proteins.
Cellular calcium regulation. Depicted are several sites that control calcium entry, efflux, and sequestration. 1. Na+, Ca++ exchange. 2. Receptor-operated channels. 3. Voltagegated channels. 4. Leak pathways. 5, 6. Entry and efflux in sarcoplasmic reticulum. 7. Plasma membrane pump. [Pg.219]

J. Benters, U. Flogel, T. Schafer, D. Leibfritz, S. Hechtenberg, D. Beyersmann, Study of the interactions of cadmium and zinc ions with cellular calcium homoeostasis using F-19-NMR spectroscopy, Biochem. J. 322 (1997) 793-799. [Pg.270]

Post et al. 1992) are also worthy of consideration. Effects on the IP3 receptor-mediated calcium release have also been shown to affect ultrarapid neuronal and cellular calcium oscillations in several different types of preparations, raising the possibility that IP3 modulation, either directly or indirectly through effects on phosphoinositide turnover, could be important to some elements of lithium s and related agents mood-stabilizing effects (Berridge 1989 Dixon et al. 1992 Lupu-Meiri et al. 1994 Varney et al. [Pg.109]

Lupu-Meiri M, Lipinsky D, Ozaki S, et al Independent external calcium entry and cellular calcium mobilization in Xenopus oocytes. Cell Calcium 16 20-28, 1994... [Pg.687]

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