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Catabolic hormone

Hyperglycemia followed by hypoglycemia Catabolic hormones release Platelet-activating factor and eicosanoid release... [Pg.319]

Catabolic hormones release Euthyroid-sick syndrome... [Pg.319]

Gluc on, secreted by the alpha cells of the islets of Langerhans, is the main catabolic hormone. [Pg.56]

I would like the reader to stop and ponder something for a few minutes. If, in theory, an athlete controlled proportionately all anabolic, androgenic, and catabolic hormones and enzymes with dominance upon protein based tissue growth, then supplied a correct ratio of amino acids, carbohydrates, and fats, the athlete would increase in protein based mass at a rate of body weight x 1.818 g daily. So a 250 pound bodybuilder in theory could add 454.5g daily in lean tissue. Though some or most would not agree, I... [Pg.187]

The breakdown of fatty acids in (3-oxidation (see Topic K2) is controlled mainly by the concentration of free fatty acids in the blood, which is, in turn, controlled by the hydrolysis rate of triacylglycerols in adipose tissue by hormone-sensitive triacylglycerol lipase. This enzyme is regulated by phosphorylation and dephosphorylation (Fig. 5) in response to hormonally controlled levels of the intracellular second messenger cAMP (see Topic E5). The catabolic hormones glucagon, epinephrine and norepinephrine bind to receptor proteins on the cell surface and increase the levels of cAMP in adipose cells through activation of adenylate cyclase (see Topic E5). The cAMP allosterically activates... [Pg.329]

It has been suggested that the catabolic response to injury may be influenced as much by the initial absence of the anabolic hormone insulin as by increases in catabolic hormones (G5). [Pg.269]

With loss of insulin action and an excess of catabolic hormones, hydrolysis of triglycerides is markedly increased, glycerol supply rises and triglyceride turnover in plasma increases with a concomitant increase in ketoacid derived from hepatic oxidation of FFA. Fatty acids are partly oxidized to ketonic compounds. Ketone synthesis increases more than threefold in the state of insulin deficiency as the result of a low insulin/glucagon ratio and a high FFA supply to the liver. At low insulin levels, ketone uptake and utilization of peripheral tissue is also significantly reduced. [Pg.8]

Catabolic hormones, c) tokines Erythropoietin synthesis, activation pf vitamin 1)... [Pg.1677]

The anabolic state is reflected by metabolite availability and the presence of anabolic or catabolic hormones. [Pg.508]

Cortisol Hydrocortisone a glucocorticoid steroid hormone synthesized by the adrenal gland in response to binding of ACTH. Cortisol binds to cytosolic or nnclear receptors that act as transcription factors for glucocorticoid-responsive genes. In general, cortisol is a catabolic hormone that promotes the breakdown of proteins. [Pg.437]

If you are training with a high level of intensity, more than one hour is counterproductive as it increases the probability of overtraining due to a catabolic hormone called cortisol. Overtraining, next to injury, is your worst enemy. Avoid it like the plague. In addition, the faster you can complete your workout, given the same amount of sets performed, the BETTER CONDITIONING obtained. [Pg.21]

Rodway, R.G. and Galbraith, H. (1979) Effects of anabolic steroids on hepatic enzymes of amino acid catabolism. Hormone and Metabolic Research, 11, 489 190. [Pg.293]

Physiological adaptations in marasmus involve the breakdown of body tissues for energy which is stimulated by catabolic hormones, such as those from the adrenal cortex, and the reduction or cessation of protein synthesis and growth in many tissues. The specific effects of the adrenal cortical hormones are the breakdown of proteins to amino acids, and conversion of amino acids to glucose for use by the brain and nervous system. [Pg.650]

The project was started with an existing model of metabolism in the cow, published and validated (Baldwin et al., 1987 referred to as Molly), which describes utilization of glucose, amino acids and fatty acids by muscle, adipose, visceral and mammary tissues at an aggregated metabolic pathway level. Elements of genetic background, response to nutritional environment and metabolic hormones are exphcitly embodied in equation forms and parameter values, such as maximal velocity, substrate sensitivity and control by anabolic and catabolic hormones. [Pg.327]


See other pages where Catabolic hormone is mentioned: [Pg.92]    [Pg.4]    [Pg.95]    [Pg.136]    [Pg.417]    [Pg.259]    [Pg.453]    [Pg.928]    [Pg.874]    [Pg.280]    [Pg.332]    [Pg.642]    [Pg.282]    [Pg.1]    [Pg.2]    [Pg.113]    [Pg.93]    [Pg.34]    [Pg.26]    [Pg.533]    [Pg.24]    [Pg.35]   
See also in sourсe #XX -- [ Pg.453 ]




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