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Beta blockers propranolol

The beta blocker propranolol (Inderal) has been studied in the treatment of impulsivity and appears to provide a slight benefit to some patients. It can be started at a dose of 10 mg taken three times per day and increased gradually to a maximum of 200mg/day. Side effects of propranolol include depression, fatigue, lowered blood pressure, impotence, and a worsening of symptoms in patients with asthma. [Pg.327]

Sedative/hypnotics zopiclone Beta-blockers propranolol, warfarin, theophylline... [Pg.93]

Mood stabilizers phenytoin, valproate, topiramate Sedative/anxiolytics diazepam, barbiturates Beta-blockers propranolol... [Pg.93]

Antihypertensives typically used in child psychiatry include a agonists (clonidine, guanfacine) and beta-blockers (propranolol, nadolol). [Pg.678]

In 1965, the beta-blocker propranolol began to be used to treat some fear or anxiety symptoms. These drugs are particularly useful for treating panic symptoms associated with social anxiety disorder. [Pg.94]

Nadolol (Corgard). Nadolol is a nonselective beta blocker that is administered orally as an antihypertensive and antianginal agent. This drug has an advantage over other nonselective beta blockers (propranolol) in that nadolol often needs to be taken only once each day. [Pg.283]

Brief History. M.R. is a 48-year-old man with a history of coronary artery disease and cardiac rhythm disturbances. Specifically, he has experienced episodes of paroxysmal supraventricular tachycardia, with his heart rate often exceeding 180 beats per minute. He has been treated for several years with the nonspecific beta blocker propranolol (Inderal). Oral propranolol (60 mg/d) has successfully diminished his episodes of tachycardia. In an effort to improve his myocardial function and overall cardiovascular fitness, M.R. recently enrolled as an outpatient in a cardiac rehabilitation program. Under the supervision of a physical therapist, he attended cardiac training sessions three times each week. A typical session consisted of warm-up calisthenics, bicycle ergometry, and cool-down stretching activities. Each session lasted approximately 45 minutes. [Pg.328]

RIZATRIPTAN BETA-BLOCKERS -PROPRANOLOL Plasma levels of rizatriptan almost doubled during propranolol therapy Propanolol inhibits the metabolism of rizatriptan Initiate therapy with 5 mg rizatriptan and do not exceed 10 mg in 24 hours. The manufacturers recommend separating doses by 2 hours, although this has not been borne out by studies... [Pg.234]

THEOPHYLLINE BETA-BLOCKERS -PROPRANOLOL t plasma levels of theophylline with propranolol Propranolol exerts a dose-dependent inhibitory effect on the metabolism of theophylline Monitor theophylline levels during propranolol co-administration... [Pg.669]

CANNABIS BETA-BLOCKERS -PROPRANOLOL 1 levels, with risk of therapeutic failure Induction of CYP1 A2-mediated metabolism by any form of smoking. Foods (e.g. broccoli, cabbage, Brussels sprouts, chargrilled meat) also induce this isoenzyme Watch for poor response to propanolol conversely, watch for toxic effects if a previously heavy cannabis user stops smoking... [Pg.696]

Other drugs that reduce theophyUine clearance, but by unknown mechanisms, are cardiac medications the beta-blocker propranolol, the calcium channel blockers... [Pg.3365]

Specific phobias generally are not treated with psychotropic medications. Social phobias often respond well to cognitive-behavioral treatment. When medications are used, the clinician has two main alternatives For occasional use—for example to reduce anxiety associated with theatrical performances or public speaking—the beta blocker propranolol has been shown to be quite effective. This drug does little to alter the cognitive aspects of anxiety (worry), but effectively reduces many physiological symptoms such as rapid heart rate. Anxious patients treated with propranolol take the medication only prior to the stressful event, ingesting 20-80 mg about one hour before. [Pg.94]

Stott PW, Williams AC, Barry BW. Mechanistic study into the enhanced transdermal permeation of a model beta-blocker, propranolol, by fatty acids A melting point depression effect. Int J Pharm 2001 219(1-2) 161-176. [Pg.407]

The P2 adrenergic agonists are used for bron-chodilatory effects in horses and may facilitate esophageal relaxation. In humans, peristaltic amplitude was increased in the distal smooth muscle part of the esophageal body after infusion of the non-selective beta blocker propranolol (Lyrenas... [Pg.95]

Answer A. Although used primarily via inhalation for asthma, systemic effects of albuterol include vasodilation due to its p2 receptor activation. This can result in a decrease in PVR and mean BP, which elicits a reflex tachycardia. Methoxamine and phenylephrine are Ct-receptor activators causing vasoconstriction, which would result in reflex bradycardia. Ganglion blockers (mecamylamine) prevent autonomic reflexes, and a reflex increase in heart rate could not occur in the presence of a beta blocker (propranolol). [Pg.79]

E. Other Drugs Other agents used in the treatment of thyrotoxicosis include the beta-blockers. Propranolol also inhibits 5 -deiodinase. These agents are particularly useful in controlling the tachycardia and other cardiac abnormalities of severe thyrotoxicosis. [Pg.339]

The client diagnosed with migraine headaches that occur every 2 to 3 days is placed on preventive therapy with the beta blocker propranolol (Inderal). Which data indicates the medication is effective ... [Pg.16]

A study of 16 patients who had been taking various beta blockers (propranolol 5, atenolol 5, metoprolol 2, bisoprolol 2, oxprenolol 1, celiprolol 1) for longer than one month found no difference in the onset and duration of action of rocuronium, when compared with a control group. Similarly, intra-operative esmolol did not affect the onset and recovery time from suxamethonium (succinylcholine) blockade in patients with normal plasma cholinesterase (pseudocholinesterase) activity, but see also (b) above. [Pg.119]

Beta Blockers propranolol, metoprolol lethargy, sleep disturbances... [Pg.70]

The method is already widely used for obtaining sol-gel electrochemical sensors. Target molecules used include molecules such as the catecholamines, dopamine [251,295], and epinephrine [302], a beta blocker, propranolol [303], and parathion and paraoxon pesticides [304]. Marx and Lion [304] reported that the selectivity for binding of parathion compared to paraoxon increased from 1.7 to 30 by the sol-gel imprinting. The insert of Figure 4.14 shows that paraoxon is merely a thio-parathion that is, the P=S bond in parathion is replaced by a P=0 bond in paraoxon, but this meager difference was sufficient to drive very large molecular selectivity. [Pg.269]

Chu LF, Cun T, Ngai LK, Kim JE, Zamora AK, Young CA, et al. Modulation of remifentanil-induced postinfusion hyperalgesia by the beta-blocker propranolol in humans. Pain May 2012 153(5) 974r-81. [Pg.116]


See other pages where Beta blockers propranolol is mentioned: [Pg.149]    [Pg.334]    [Pg.761]    [Pg.334]    [Pg.93]    [Pg.390]    [Pg.860]    [Pg.36]    [Pg.661]    [Pg.463]    [Pg.168]    [Pg.94]    [Pg.102]    [Pg.102]    [Pg.151]    [Pg.68]    [Pg.393]    [Pg.11]    [Pg.481]    [Pg.482]    [Pg.2606]    [Pg.99]    [Pg.179]    [Pg.280]   
See also in sourсe #XX -- [ Pg.16 ]




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