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Benign recurrent cholestasi

Is an acute disturbance in hepatic transport of bile acids the primary cause of cholestasis in benign recurrent cholestasis Lancet, 1 1249 (1974). [Pg.233]

G. F. de Pagter, Familial and nonfamilial benign recurrent cholestasis distinguished by plasma disappearance of indocyanine green but not cholyglycerine. Gut, 19 345 (1978). [Pg.234]

F. Successful chnical apphcation of extracorporal albumine dialysis in a patient with benign recurrent intrahepatic cholestasis (BRIC). Z. Gastroenterol. 2001 39 (Suppl. 2) 13-14... [Pg.242]

G. Benign recurrent intrahepatic cholestasis. A clinico-pathologic study. Res. Clin. Lab. 1990 20 19-27... [Pg.242]

Nakamuta, M., Sakamoto, S., Miyata, Y., Sato, M., Nawata, H. Benign recurrent intrahepatic cholestasis a long-term follow-up. Hepat. Gastroenterol. 1994 41 287 - 289... [Pg.242]

Sturm, E., Franssen, CJtJM., Gouw, A., Staels, B., Boverhof, R., de Knegt, R.J., Stellaard, F., Biileveld, C.MA., Kuipers, F. Extracorporal albumin dialysis (MARS) improves cholestasis and normalizes low apo A-I levels in a patient with benign recurrent intrahepatic cholestasis (BRIC). Liver 2002 22 (Suppl. 2), 72-75... [Pg.242]

Cystine storage disease Gaucher s disease Glycogen stora ge disease Juvenile, Tay-Sachs disease Niemann-Pick disease Tay-Sachs disease Chronic renal failure Hemolytic-uremic syndrome Nephrotic syndrome Benign recurrent intrahepatic cholestasis... [Pg.928]

Familial diseases include Dubin-Johnson syndrome, Rotor s syndrome, and benign familial recurrent cholestasis. Serum bilirubin values in Dubin-Johnson syndrome are the same as those found in Rotor s syndrome (Table 29-2). Very little is known about benign familial recurrent cholestasis. All three disorders are uncommon or rare, and all are benign. The liver in Dubin-Johnson... [Pg.695]

ATP8B1 NBD-PS, PS , PE Progressive familial intrahepatic cholestasis, benign recurrent intrahepatic cholestasis... [Pg.458]

The remaining 59 patients, with relatively low serum bile acid concentrations (SGLC = 2.2 0.3,GC = 2.2 0.3, 3a-hydroxy bile acids = 10.3 0.9), included patients with mild liver disease, intestinal dysfunctions such as Crohn s disease, coeliac disease and gastroenteritis, acute lymphatic leukemia treated with methotrexate, contaminated small bowel syndrome and three patients with benign recurrent intrahepatic cholestasis (BRIC). [Pg.226]

Serum concentration of sulfated lithocholic acid conjugates (SGLC) and glycocholic acid (GC) in two brothers with benign recurrent intrahepatic cholestasis after oral fat loading at t = 0 hours A. during a symptom-free period B. as A, cholestyramine added C. during a pre-icteric period. [Pg.229]

Huinink and K. H. Brandt, Familial benign recurrent intrahepatic cholestasis Interrelation with intrahepatic cholestasis of pregnancy and from oral contraceptives, Gastroenterol., 71 202 (1976). [Pg.233]


See other pages where Benign recurrent cholestasi is mentioned: [Pg.242]    [Pg.604]    [Pg.242]    [Pg.242]    [Pg.604]    [Pg.242]    [Pg.56]    [Pg.459]    [Pg.281]    [Pg.283]    [Pg.223]    [Pg.227]    [Pg.231]    [Pg.233]    [Pg.233]    [Pg.242]    [Pg.242]    [Pg.417]    [Pg.299]    [Pg.302]    [Pg.456]    [Pg.102]    [Pg.232]    [Pg.234]    [Pg.858]   
See also in sourсe #XX -- [ Pg.233 ]




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Benign

Benign familial recurrent cholestasis

Cholestasis

Recurrence

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