Atrophic thyroiditis

In the baseline study, the prevalence of autoimmune thyroiditis (AIT) in Zhangwu (MAI) and Huanghua (El) was 3.8 and 6.2 times, respectively, compared with the prevalence of AIT in Panshan (MDI). The prevalence of Hashimoto s thyroiditis in MAI and El was 2.8 and 4.1 times, respectively, compared with that of Hashimotos thyroiditis in MDI. The prevalence of atrophic thyroiditis in MAI and El was 7.7 and 14.4 times, respectively, compared with that in MDI (Table 125.2). The cumulative incidence of AIT in MAI and MDI was 4.4 and 5.5 times, respectively, compared with the cumulative incidence of AIT in MDI (Table 125.2). Both the cross-sectional and the prospective study confirmed that the prevalence and incidence of AIT increased as iodine intake rose. 

Jonckheer M.H., Coomans D., Massart D.L., Vanhaelst L., Deconinck F. 1982. The application of SIMCA pattern recognition to the study of autoimmune atrophic thyroiditis. Nucl.Med.Com. 3 70-77. 

Addisons disease, results from progressive destruction or dysfunction of the adrenal glands by a local disease process or systemic disorder (Box 51-12), Worldwide, infectious diseases are the most common cause of primary adrenal insufficiency and include tuberculosis, fongal infections (histoplasmosis and cryptococcosis), and cytomegalovirus infections. Autoimmune adrenalitis accounts for more than 70% of cases reported in the Western world, with adrenal autoantibodies noted in more than 75% of cases,. The adrenal glands are atrophic, with loss of cortical cells but an intact medulla. Almost 50% of patients with autoimmune adrenalitis have an associated autoimmune disease with autoimmune thyroid disease the most common. 

Primary myxoedema. Atrophic form of diffuse autoimmune thyroiditis. See also thyroiditis, autoimmune. 

Atrophic Autoimmune Thyroiditis relationship between the clinical state and intrathyroidal iodine as measured in vivo in man. J. Clin. Endocrinol. Metab. 

Evidence has been published suggesting that the progressively decreasing intrathyroidal stores parallel and probably condition the incidious development of hypothyroidism in atrophic autoimmune thyroiditis (17, 18). XRF measurements have been helpful in our hands (11) when the results were found to be low, to stimulate more thorough investigations than would have been felt otherwise necessary to come to a definite diagnosis and also to decide whether or not substitution should be started,. 

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