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Asthma reducing

Long-term control of Bronchial asthma, reduces need for oral corticosteriod therapy for asthma Oral Inhalation 40-160 meg twice a day. Maximum 320 meg twice a day. Rhinitis, prevention of recurrence of nasal polyps Nasal Inhalation 1 spray in each nostril 2-4 times a day or 2 sprays twice a day. Maintenance 1 spray 3 times a day. [Pg.120]

Bronchial constriction, especially in patients with asthma Reduced pulmonary blood flow and increased pulmonary blood pressure... [Pg.2956]

Isocyanates spasms of the larynx, pulmonary edema Cough, dyspnea Asthma, reduced pulmonary... [Pg.32]

Forced vital capacity (FVC) quantifies the maximum air volume expired following a maximal inspiration and is one of the basic measures of analyzing flow changes such as reduced airway patency observed in asthma. To measure FVC, an individual inhales maximally and then exhales as rapidly and completely as possible. FVC primarily reflects the elastic properties of the respiratory tract. The gas volume forcibly expired within a given time interval, FEV (where t is typically one second, FEVj q)... [Pg.210]

For children, Uie new standard will reduce respiratory problems, such as asthma tUtacks. It will result in one million fewer incidences of decreased lung function in cliildren each year... [Pg.36]

Platelet activating factor (PAF) was first identified by its ability (at low levels) to cause platelet aggregation and dilation of blood vessels, but it is now known to be a potent mediator in inflammation, allergic responses, and shock. PAF effects are observed at tissue concentrations as low as 10 M. PAF causes a dramatic inflammation of air passages and induces asthma-like symptoms in laboratory animals. Toxic-shock syndrome occurs when fragments of destroyed bacteria act as toxins and induce the synthesis of PAF. This results in a drop in blood pressure and a reduced... [Pg.247]

Daily asthma attacks, often by night FEV1 <60% reduced physical stamina daily use of bronchodilators. Rapid-acting inhaled p2-agonist Inhaled glucocorticoid... [Pg.288]

Asthma is a chronic inflammatory disease. Therefore steroids represent the most important and most frequently used medication. Already after the fust treatment, steroids reduce cellular infiltration, inflammation, and the LAR, whereas changes in the EAR require prolonged treatment to lower the existent IgE levels. The mechanisms of steroid actions are complex and only incompletely understood. Besides their general antiinflammatory properties (see chapter glucocorticoids), the reduction of IL-4 and IL-5 production from T-lymphocytes is particularly important for asthma therapy. The introduction of inhaled steroids, which have dramatically limited side effects of steroids, is considered one of the most important advancements in asthma therapy. Inhaled steroids (beclomethasone, budesonide, fluticasone, triamcinolone, momethasone) are used in mild, moderate, and partially also in severe asthma oral steroids are used only in severe asthma and the treatment of status asthmaticus. Minor side effects of most inhaled steroids are hoarseness and candidasis, which are avoided by the prodrug steroid ciclesonide. [Pg.289]

Inhaled steroids (commonly used are beclomethasone, budesonide, triamcinolone, fluticasone, flunisolide) appear to attenuate the inflammatory response, to reduce bronchial hyperreactivity, to decrease exacerbations and to improve health status they may also reduce the risk of myocar dial infar ction, but they do not modify the longterm decline in lung function. Whether- steroids affect mortality remains unclear. Many patients appear to be resistant to steroids and large, long-term trials have shown only limited effectiveness of inhaled corticosteroid ther apy. Certainly, the benefit from steroids is smaller in COPD than in asthma. Topical side-effects of inhaled steroids are oropharyngeal candidiasis and hoarse voice. At the normal doses systemic side-effects of inhaled steroids have not been firmly established. The current recommendation is that the addition of inhaled gluco-coiticosteroids to bronchodilator treatment is appropriate for patients with severe to veiy sever e COPD. [Pg.365]

Maintenance and prophylactic treatment of asthma for asthma patients who require systemic corticosteroid administration when adding an inhaled corticosteroid may reduce or eliminate the need for systemic corticosteroids... [Pg.339]

Respiratory acidosis results when decreased respiration raises the concentration of C02 in the blood. Asthma, pneumonia, emphysema, or inhaling smoke can all cause respiratory acidosis. So can any condition that reduces a person s ability to breathe. Respiratory acidosis is usually treated with a mechanical ventilator, to assist the victim s breathing. The improved exhalation increases the excretion of C02 and raises blood pH. In many cases of asthma, chemicals can facilitate respiration by opening constricted bronchial passages. [Pg.573]


See other pages where Asthma reducing is mentioned: [Pg.1433]    [Pg.717]    [Pg.1433]    [Pg.717]    [Pg.255]    [Pg.436]    [Pg.441]    [Pg.443]    [Pg.445]    [Pg.181]    [Pg.114]    [Pg.359]    [Pg.110]    [Pg.200]    [Pg.216]    [Pg.295]    [Pg.141]    [Pg.48]    [Pg.101]    [Pg.287]    [Pg.289]    [Pg.296]    [Pg.364]    [Pg.541]    [Pg.543]    [Pg.546]    [Pg.688]    [Pg.798]    [Pg.888]    [Pg.1053]    [Pg.347]    [Pg.347]    [Pg.72]    [Pg.30]    [Pg.122]    [Pg.580]    [Pg.196]    [Pg.169]    [Pg.108]    [Pg.221]    [Pg.224]    [Pg.224]   
See also in sourсe #XX -- [ Pg.442 ]




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