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Amphotericin B, nephrotoxicity

Amphotericin B nephrotoxicity can be reduced by slowing the infusion rate to 24 hours or, in at-risk patients, substituting liposomal amphotericin B. [Pg.867]

Deray G. Amphotericin B nephrotoxicity. J Antimicrob Chemother. 2002 49(suppl 1) 37—4-1. [Pg.561]

Goldman RD, Koren G. Amphotericin B nephrotoxicity in children. J Pediatr Hematol Oncol. 2004 26 421-426. [Pg.562]

Vita E, Schroeder DJ. Intralipid in prophylaxis of amphotericin B nephrotoxicity. Ann Pharmacother 1994 28(10) 1182-3. [Pg.206]

Llanos A, Cieza J, Bernardo J, Echevarria J, Biaggioni 1, Sabra R, Branch RA. Effect of salt snpplementation on amphotericin B nephrotoxicity. Kidney Int 1991 40(2) 302-8. [Pg.210]

Anderson CM. Sodium chloride treatment of amphotericin B nephrotoxicity. Standard of care West J Med 1995 162(4) 313-17. [Pg.210]

Heidemann HT, Gerkens JF, Spickard WA, Jackson EK, Branch RA. Amphotericin B nephrotoxicity in humans decreased by salt repletion. Am J Med 1983 75(3) 476-81. [Pg.210]

Camp MJ, Wingard JR, Gilmore CE, Lin LS, Dix SP, Davidson TG, Geller RB. Efficacy of low-dose dopamine in preventing amphotericin B nephrotoxicity in bone marrow transplant patients and leukemia patients. Antimicrob Agents Chemother 1998 42(12) 3103-6. [Pg.210]

Goren MP, Viar MJ, Shenep JL, Wright RK, Baker DK, Kalwinsky DK. Monitoring serum aminoglycoside concentrations in children with amphotericin B nephrotoxicity. Pediatr Infect Dis J 1988 7(10) 698-703. [Pg.210]

Deray G. 2002 Feb. Amphotericin B nephrotoxicity.TAnf/m/crobC/remof/rer49 Supp11 37-41. [Pg.42]

Branch RA, Jackson EK, Jacqz E, et al. Amphotericin-B nephrotoxicity in humans decreased by sodium supplements with coadministration ofticarcillin or intravenous saline. Klin Wochenschr 1987 65 500-6. [Pg.345]

Wasan KM, Conklin JS. Enhanced amphotericin B nephrotoxicity in intensive care patients with elevated levels of low-density lipoprotein cholesterol. Clin Infect DIs 1997 24 78-80. [Pg.345]

Gouge TH, Andriole VT. An experimental model of amphotericin B nephrotoxicity with renal tubular acidosis. J Lab Clin Med 1971 78 713-24. [Pg.346]

Cheng JT, Witty RT, Robinson RR,Yarger WE. Amphotericin B nephrotoxicity increased renai resistance and tubuie permeabiiity. Kidney int 1982 22 626-33. [Pg.347]

Sabra R, Zeinoun N, Sharaf LH, et al. Role of humoral mediators in, and influence of a liposomal formulation on, acute amphotericin B nephrotoxicity. PharmacoiToxicoi 2001 88 168-75. [Pg.347]

Gerkens JF, Branch RA. The influence of sodium status and furosemide on canine acute amphotericin B nephrotoxicity. J Pharmacol Exp Ther 1980 214 306-11. [Pg.347]

Ohnishi A, Ohnishi T, Stevenhead W, et al. Sodium status influences chronic amphotericin B nephrotoxicity in rats. Antimicrob Agents Chemother 1989 33 1222-7. [Pg.347]

Tolins JP, Raij L. Chronic amphotericin B nephrotoxicity in the rat protective effect of calcium channel blockade. J Am Soc Nephrol 1991 2 98-102. [Pg.347]

Heidemann HT, Bolten M, Inselmann G. Effect of chronic theophylline administration on amphotericin B nephrotoxicity in rats. Nephron 1991 59 294-8. [Pg.347]

Luke DR, Wasan KM, Verani RR, et al. Attenuation of amphotericin-B nephrotoxicity in the candidiasis rat model. Nephron 1991 59 139-44. [Pg.348]

Kuan CJ, Branch RA, Jackson EK. Effect of an adenosine receptor antagonist on acute amphotericin B nephrotoxicity. Eur J Pharmacol 1990 178 285-91. [Pg.348]

Rosch JM, Pazin GJ, Fireman P. Reduction of amphotericin B nephrotoxicity with mannitol. JAMA 1976 235 1995-6. [Pg.348]

Major functions of the distal nephron include the regeneration of bicarbonate, the excretion of acid (hydrogen ion), the secretion of potassium, and the reabsorption of water. Damage to this portion of the nephron may present as significant acidemia and either hypo-or hyperkalemia, depending on the mechanism of injury. For example, amphotericin B produces small pores in the luminal membrane of distal tubular cells. These pores allow small molecules such as potassium to leak out the molecules are then wasted in the urine. Consequently, amphotericin B nephrotoxicity is characterized by hypokalemia secondary to renal potassium wasting. ATN is associated with urinary sediment characterized by the presence of tubular cells, coarse granular casts, and rarely, RBC casts. [Pg.786]

Bullock WE, Luke RG, Nuttal CE, Bhathena D. Can mannitol reduce amphotericin B nephrotoxicity Double blind study and description of a new vascular lesion in kidneys. Antimicrob Agents Chemother 1976 10 555-563. [Pg.217]

Takacs FJ, Tomkiewicz ZM, Merrill JR Amphotericin B nephrotoxicity with irreversible renal failure. Ann Intern Med 1963 59 716-724. [Pg.218]

See other pages where Amphotericin B, nephrotoxicity is mentioned: [Pg.209]    [Pg.345]    [Pg.348]    [Pg.348]    [Pg.348]    [Pg.1485]    [Pg.877]    [Pg.889]    [Pg.217]   
See also in sourсe #XX -- [ Pg.565 ]



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