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Acute hepatitis follow

Mazzanti G, BattineUi L, Daniele C, Mastroianni CM, Lichtner M, Coletta S, Costantini S. New case of acute hepatitis following the consumption of Shou Wu Pian, a Chinese herbal product derived from Polygonum multiflorum. Ann Intern Med 2004 140(7) W30. [Pg.2890]

A 62-year-old woman with no history of liver disease developed an episode of self-limited acute hepatitis following consumption of 2 liters of noni juice over a 3-month period. A percutaneous liver biopsy revealed acute hepatitis consistent with an idiosyncratic drug reaction. Elevated levels of AST, ALT, and bilirubin were observed and returned to normal after cessation of noni. The woman had been treated the previous year with fludarabine for chronic B-cell leukemia (Stadlbauer et al. 2005). [Pg.576]

Roda G, Caponi A, Belluzzi A, Roda E. Severe cholestatic acute hepatitis following azathioprine therapy in a patient with ulcerative pancolitis. Dig Liver Dis 2009 41(12) 914-5. [Pg.838]

Death. 1,2-Dibromoethane can be fatal to humans after oral or dermal exposure. Acute deaths following toxic doses are related to cardiopulmonary arrest or, if affected individuals survive for a period of time, to hepatic and renal failure. These results are supported by animal studies in which acute death occurred after oral, dermal, and inhalation exposure. [Pg.58]

The frequency of chronic hepatitis following acute HBV infection is 5-10%. The total number of patients with chronic infection B is estimated at 350-400 million worldwide, in Germany about 500,000. HBV infection in childhood leads to a higher rate of chronicity. Lethality rate is up to 1%. (see chapter 22.4 )... [Pg.699]

The hepatopulmonary syndrome (HPS) may occur in patients with chronic liver disease or in the course of acute hepatic failure. It is characterized by the following criteria (7.) exclusion of an underlying pulmonary or cardiac disease, (2.) increase in the alveolocapillary oxy-... [Pg.734]

Katsushima, S., Inokuma, T., Oi, H., Okamura, J., Higashi, T., Takeuchi, R., Hidaka, A., Shigeno, C, lida, Y., Konishi, J. Acute hepatic failure following transcatheter arterial embolization for the treatment of hepatocellular carcinoma. Digestion 1997 58 189-195... [Pg.803]

Centers for Disease Control and Prevention (CDC). Acute hepatitis and renal failure following ingestion of raw carp gallbladders—Maryland and Peimsylvania, 1991 and 1994. MMWR Morb Mortal Wkly Rep 1995 44(30) 565-6. [Pg.241]

Six cases of acute hepatitis occurred in four patients during the first 10 weeks of treatment, and in two after 15 weeks of therapy (3). They were represented by jaundice (n = 5), itching (n = 2), weakness (n = 3), and anorexia, nausea, and vomiting (n = 2). Increases in liver enzymes varied from 1.5 to over 30 times the upper limit of the reference range. Liver biopsy showed centrilobular or panlobular bridging necrosis in the four women and intra-hepatic cholestasis in the two men. Complete recovery of normal liver function tests ensued at follow-up after interruption of nimesulide therapy. [Pg.2524]

A previously healthy 46-year-old man developed acute fulminant hepatitis following treatment with rabepra-zole, citalopram hydrobromide, terbinafine, and a multivitamin formulation (41). Liver biopsy showed submassive centrilobular necrosis and intrahepatic cholestasis with florid bile duct proliferation. [Pg.3317]

Five workers at an industrial waste treatment plant in Ulsan, Korea, developed acute toxic hepatitis following the introduction of a new disposal process that resulted in their exposure to a large number of lipophilic and hydrophilic volatile organic compounds. The chemicals they were exposed to included... [Pg.502]

Hepatitis is a major cause of morbidity and mortality in the United States. Viral hepatitis refers to the clinically important hepatotrophic viruses responsible for hepatitis A (HAV), hepatitis B (HBV), hepatitis C (HCV), delta hepatitis, and hepatitis E. Hepatitis G vims has also been described however, its role in chnical illness is stiU not clear. Viral hepatitis has acute, fulminant, and chronic clinical forms, defined by duration or severity of infection. The clinical, hiochemi-cal, immunoserologic, and histologic features of viral hepatitis follow similar patterns regardless of the virus responsible for the patient s illness. Hepatocellular response to injury and the resulting physical signs and symptoms are nonspecific. [Pg.737]

Wiegand J, Jackel E, Cornberg M et al. (2004) Long-term follow-up after successful interferon therapy of acute hepatitis C. Hepatology 40 98-107... [Pg.289]

Cases of acute hepatitis have been reported as follows a woman who had taken 2 tablets daily of shen-min tablets (20% 12 1 extract of fo-ti and 40% fo-ti powder) for 8 weeks (Cardenas et al. 2006) a man who had taken fo-ti tablets at the recommended dose daily for 1 month (Battinelli et al. 2004) and a woman who had taken fo-ti tablets for 2 weeks... [Pg.730]


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See also in sourсe #XX -- [ Pg.1808 ]




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