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Toxicosis from

Pokras, M.A. and R. Chafel. 1992. Lead toxicosis from ingested fishing sinkers in adult common loons (Gavia immer) in New England. Jour. Zoo Wildl. Med. 23 92-97. [Pg.339]

HV160 Puls R., and J. A. Greenway. Fusario-toxicosis from barley in British Columbia. II. Analysis and toxicity of syspected barley. Can J Comp Med 1976 40(1) 16-19. [Pg.258]

Bose, R., Pinsky, C., and Glavin, G.B., Sensitive murine model and putative antidotes for behavioural toxicosis from contaminated mussel extracts. Can. Dis. WHy Rep., 16(Suppl IE), 91, 1990. [Pg.429]

CompIkralifHis. Therapy is usually discontinued after 5 days to avoid complications or toxicosis from calcium EDTA therapy. [Pg.198]

Phytonadione (vitamin K.) is the antirfotefor toxicosis from an anticoagulwt rodenticitie (e.g., diphacinone). Phytonadione supplies additiotial reduced vitamin K to replace dial which is lost when the coumarin and inilane-dione andcoagulants interfere with vitamin K epoxide reductase in the synthesis of coagula-donfacmrs. [Pg.295]

Diagnosis of toxicosis from marijuana (and other illicN drugs) is problematic because histories may be incomplete or misleading. [Pg.310]

Exposure. Toxicosis from ingesting antifreeze is a relalively common occunence in dogs... [Pg.316]

Toxicity Consumption of seeds at 0.1 %-0.3% of body weight niay be toxics whereas toxicosis from green foliage requires dosages equalling 10%-20% of body weight. [Pg.372]

D. I Toxidty. Voluntary feed refusal often prevents full expression of oral toxicosis from conta-minated feeds. Fiowever, iJosing studies using parenteral injection and oral administration reveal that trichothecenes are potent toxicants. [Pg.420]

The answer is 1 [Chapter 26 VIVB 1-2,4]. Acidic rumen conditions supply protons arid maintain the ammonia released from urea In an ionized state (NH "), which increases its solubility in water and retards Its absorption and transport across epithelium. This is a form of ion trapping, and is, in part, why acidification of the rumen with acetic acid or vinegar helps to alleviate ammonia toxicosis from urea. All other statements are true statements arvl reinforce the important points that ammo-nia toxicosis results in metabolic acidosis, is most likely to cKCur in fasting animals that are unaccustomed to urea, arxl is a rapidly progressing acute to peracute disease. Proper preservation of samples is necessary to prevent changes in ammxiia concentrations. [Pg.464]

Outbreaks of poisoning from the ingestion of mycotoxins occur frequently in domestic animals, because they occasionally consume moldy feeds in bulk. In contrast, acute exogenic toxicosis from mycotoxins in humans is relatively uncommon, for humans usually refuse moldy food. Therefore, the deleterious effects, including cancer, caused by the chronic consumption of mycotoxins are of more importance to man than acute poisoning. Carcinogenic mycotoxins have thus attracted the primary interest of many investigators. However, the importance of neurotropic mycotoxins should also be emphasized because of their violent and sometimes lethal effects in cases of acute mycotoxicoses. [Pg.193]


See other pages where Toxicosis from is mentioned: [Pg.236]    [Pg.253]    [Pg.236]    [Pg.253]    [Pg.372]    [Pg.384]    [Pg.233]    [Pg.295]    [Pg.332]    [Pg.335]    [Pg.632]   
See also in sourсe #XX -- [ Pg.62 , Pg.205 , Pg.252 , Pg.254 , Pg.295 , Pg.296 , Pg.314 , Pg.316 , Pg.329 , Pg.331 , Pg.347 , Pg.348 , Pg.349 , Pg.386 , Pg.387 , Pg.388 , Pg.446 , Pg.456 , Pg.460 , Pg.468 ]




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