Toxicity sublethal effects

Thus, there is not a great deal of difference between the three classes in acute toxicity all are highly toxic. However, methyl mercury is more persistent than the other two types, and so has the greater potential to cause chronic toxicity. The latter point is important when considering the possibility of sublethal effects. 

Moriarty, F.M. (1968). The toxicity and sublethal effects of p-p -DDT and dieldrin to Aglais urticae and Chorthippus brunneus. Annals of Applied Biology 62, 371-393. 

Calabrese A, Thurberg FP, Dawson MA, WenzlofF DR. 1975. Sublethal physiological stress induced by cadmium and mercury in the winter flounder (Pseudoplumnectes amer-icanus). In Koeman JH, Strik JJ, editors, Sublethal effects of toxic chemicals on aquatic animals. Amsterdam Elsevier. 

Aniline may be absorbed following inhalation, ingestion, and dermal exposures. The inhalation toxicity of aniline was studied in several animal species, but only one study that utilized multiple exposure concentrations for sublethal effects was located. Data from human studies lack specific details or exposures... 

Calabrese, A., F.R Thurberg, M.A. Dawson, and D.R. Wenzloff. 1975. Sublethal physiological stress induced by cadmium and mercury in winter flounder, Pseudopleuronectes americanus. Pages 15-21 in J.H. Koeman and J.J.T.W.A. Strik (eds.). Sublethal Effects of Toxic Chemicals on Aquatic Animals, Elsevier Sci. Publ. Co., Amsterdam. 

No data were available on toxic or sublethal effects of lead to reptiles under controlled conditions. 

Pentachlorophenol was most toxic and most rapidly metabolized in aquatic environments at elevated temperatures and reduced pH. Adverse effects on growth, survival, and reproduction of representative sensitive species of aquatic organisms occurred at PCP concentrations of about 8 to 80 pg/L for algae and macrophytes, about 3 to 100 pg/L for invertebrates (especially molluscs), and <1 to 68 pg/L for fishes, especially salmonids. Fatal PCP doses for birds were 380 to 504 mg/kg BW (acute oral), >3850 mg/kg in diets, and >285 mg/kg in nesting materials. Adverse sublethal effects were noted at dietary levels as low as 1.0 mg/kg ration. Residues (mg/kg fresh weight) in birds found dead from PCP poisoning were >11 in brain, >20 in kidney, >46 in liver, and 50 to 100 in egg. 

For avian wildlife, data are incomplete on PAH background concentrations and on acute and chronic toxicity. Studies with mallard embryos and PAHs applied to the egg surface showed toxic and adverse sublethal effects at concentrations between 0.036 and 0.18 pg PAH/kg whole egg (Hoffman and Gay 1981). Additional research is needed on petroleum-derived PAHs and then-effects on developing embryos of seabirds and other waterfowl. There is an urgent need for specific avian biomarkers of PAH exposure (Murk et al. 1996). 

---