Tachycardia caused

Propranolol is indicated in the management of a variety of cardiac rhythm abnormalities that are totally or partially due to enhanced adrenergic stimulation. In selected cases of sinus tachycardia caused by anxiety, pheochromocytoma, or thyrotoxicosis, (3-blockade will reduce the spontaneous heart rate. 

These drugs may cause excessive vasodilation as evidenced by swelling in the feet and ankles, and some patients may also experience orthostatic hypotension. Abnormalities in heart rate (too fast, too slow, irregular) may also occur, and reflex tachycardia—caused by excessive peripheral vasodilation—has been noted... 

SOTALOL DIURETICS-CARBONIC ANHYDRASE INHIBITORS, LOOP DIURETICS, THIAZIDES t risk of ventricular arrhythmias, particularly torsades de pointes ventricular tachycardia, caused by sotalol Hypokalaemia, a side-effect of these diuretics, predisposes to arrhythmias during sotalol therapy Normalize potassium levels before starting sotalol in patients already taking these diuretics. When starting these diuretics in patients already taking sotalol, monitor potassium levels eveiy 4-6 weeks until stable... 

The heart may be affected by both muscarinic and nicotinic effects. In the former, stimulation of the parasympathetic nerve endings, while in the latter, excess ACh on the nicotinic receptors, is of importance. The cardiovascular effects are tachycardia caused by the overstimulation of the sympathetic system, bradyarrhythmias, atrioventricular block, hypotension and QT prolongation, VF, and TdP (Grmec et al, 2004). 

Effective prophylactically in angina of effort (not vasospastic) and offset reflex tachycardia caused by nitrates. 

A) Decreased blood pressure caused by hexamethonium Increased blood pressure caused by nicotine Increased skeletal muscle strength caused by neostigmine Tachycardia caused by exercise Tachycardia caused by infusion of acetylcholine Which of the following best describes the mechanism of action of scopolamine ... 

SC-30552 (42) given orally to SHR (spontaneously hypertensive rats) lowered blood pressure and caused bradycardia. Tachycardia caused by isoproterenol was not affected by SC-30552, whereas isoproterenol-induced decreases in blood pressure were attenuated. Ganglionic blockade caused by hexamethonium did not affect vasodilatation caused by this CEB. A pA2 value against calcium-induced contractions of rabbit, aortic strips of 7.42 was calculated. These effects suggest possesses some but not 6i- or a-adrenoceptor activity and may depress the SA node. 

A. Cardiotoxic effects of the type la agents include sinus bradycardia sinus node arrest or asystole PR, QRS, or QT interval prolongation sinus tachycardia (caused by anticholinergic effects) polymorphous ventricular tachycardia (torsade de pointes) and depressed myocardial contractility, which, along with alpha-adrenergic or ganglionic blockade, may result in hypotension and occasionally pulmonary edema. 

B. Reversal of hypotension and tachycardia caused by excessive beta-adrenergic activity resulting from theophylline or caffeine overdose. 

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