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Sendai virus hemagglutination

In a recent paper (3l) it was demonstrated that the removal of sialic acid from human erythrocytes with Vibrio oholerae sialidase abolished hemagglutination by Sendai virus. Hemagglutination titers were restored selectively by the incorporation of NeuAc with 8-galactoside a2-+3sialyltransferase which has a strict... [Pg.386]

Y. Yonemitsu, Y. Kaneda, R. Morishita, K. Nakagawa, Y. Nakashima, and K. Sueishi, Characterization of in vivo gene transfer into the arterial wall mediated by the Sendai Virus (Hemagglutinating Virus of Japan) liposomes An effective tool for the in vivo study of arterial diseases, Laboratory Investigation 75 313 (1996). [Pg.264]

Sendai virus, like other myxo- and paramyxovirus, has surface glycoprotein spikes which adsorb to specific receptors on erythrocytes of most mammalian and fowl species and cause hemagglutination. The receptors on erythrocyte membranes contain neuraminic acid, as indicated by the fact that they are destroyed by neuraminidase. Haywood (3 ) demonstrated that liposomes containing gangliosides could inhibit the agglutination of erythro-... [Pg.383]

Hemagglutinating virus of Japan (HVJ Sendai virus) with liposomes... [Pg.166]

Y. Suzuki, M. Suzuki, M. Matsunaga, and M. Matsumoto, Gangliosides as paramyxovirus receptor. Structural requirement of sialo-oligosaccharides in receptors for hemagglutinating virus of Japan (Sendai virus) and Newcastle disease virus,/. Biochem. (Tokyo), 97 (1985) 1189-1199. [Pg.470]

Suzuki, Y., Suzuki, T., and Matsumoto, M., 1983, Isolation and characterization of receptor sialoglycoprotein for hemagglutinating virus of Japan (Sendai virus) from bovine erythrocyte membranes, J. Biochem. 93 1621-1633. [Pg.335]


See other pages where Sendai virus hemagglutination is mentioned: [Pg.384]    [Pg.50]    [Pg.36]    [Pg.384]    [Pg.50]    [Pg.36]    [Pg.371]    [Pg.266]    [Pg.266]    [Pg.315]    [Pg.327]    [Pg.328]    [Pg.327]    [Pg.317]    [Pg.321]   
See also in sourсe #XX -- [ Pg.386 ]




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